Judith J. Prochaska

Dr. Judith J. Prochaska

Stanford Prevention Research Center, Department of Medicine, School of Medicine, Stanford University, Stanford, CA 94305, USA
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Research Focus & Metrics

Academic Career Length

28Years (1999-Present)

(Calculated once Scilit publication data is synced: current year − year of first publication + 1)

Representative Publications

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1 May 2000 in Medicine & Science in Sports & Exercise
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Journal Article
Closed access
JSJames Sallis
James Sallis
Judith J. ProchaskaJudith J. Prochaska
Judith J. Prochaska
WTWendell C. Taylor
Wendell C. Taylor
SALLIS, J. F., J. J. PROCHASKA, and W. C. TAYLOR. A review of correlates of physical activity of children and adolescents. Med. Sci. Sports Exerc., Vol. 32, No. 5, pp. 963–975, 2000. Background Understanding the factors that influence physical activity can aid the design of more effective interventions. Previous reviews of correlates of youth physical activity have produced conflicting results. Methods A comprehensive review of correlates of physical activity was conducted, and semiquantitative results were summarized separately for children (ages 3–12) and adolescents (ages 13–18). The 108 studies evaluated 40 variables for children and 48 variables for adolescents. Results About 60% of all reported associations with physical activity were statistically significant. Variables that were consistently associated with children’s physical activity were sex (male), parental overweight status, physical activity preferences, intention to be active, perceived barriers (inverse), previous physical activity, healthy diet, program/facility access, and time spent outdoors. Variables that were consistently associated with adolescents’ physical activity were sex (male), ethnicity (white), age (inverse), perceived activity competence, intentions, depression (inverse), previous physical activity, community sports, sensation seeking, sedentary after school and on weekends (inverse), parent support, support from others, sibling physical activity, direct help from parents, and opportunities to exercise. Conclusion These consistently related variables should be confirmed in prospective studies, and interventions to improve the modifiable variables should be developed and evaluated.

1 May 2001 in Archives of Pediatrics & Adolescent Medicine
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Journal Article
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Judith J. ProchaskaJudith J. Prochaska
Judith J. Prochaska
JSJames Sallis
James Sallis
BLBarbara Long
Barbara Long
Objective To develop a reliable and valid physical activity screening measure for use with adolescents in primary care settings. Study Design We conducted 2 studies to evaluate the test-retest reliability and concurrent validity of 6 single-item and 3 composite measures of physical activity. Modifications were based on the findings of the 2 studies, and a best measure was evaluated in study 3. Accelerometer data served as the criterion standard for tests of validity. Results In study 1 (N = 250; mean age, 15 years; 56% female; 36% white), reports on the composite measures were most reliable. In study 2 (N = 57; mean age, 14 years; 65% female; 37% white), 6 of the 9 screening measures correlated significantly with accelerometer data. Subjects, however, had great difficulty reporting bouts of activity and distinguishing between intensity levels. Instead, we developed a single measure assessing accumulation of 60 minutes of moderate to vigorous physical activity. Evaluated in study 3 (N = 148; mean age, 12 years; 65% female; 27% white), the measure was reliable (intraclass correlation, 0.77) and correlated significantly (r= 0.40,P<.001) with accelerometer data. Correct classification (63%), sensitivity (71%), and false-positive rates (40%) were reasonable. Conclusion The "moderate to vigorous physical activity" screening measure is recommended for clinical practice with adolescents.

1 December 2004 in Journal of Consulting and Clinical Psychology
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Journal Article
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Judith J. ProchaskaJudith J. Prochaska
Judith J. Prochaska
KDKevin Delucchi
Kevin Delucchi
SHSharon M. Hall
Sharon M. Hall
This meta-analysis examined outcomes of smoking cessation interventions evaluated in 19 randomized controlled trials with individuals in current addictions treatment or recovery. Smoking and substance use outcomes at posttreatment and long-term follow-up (> or = 6 months) were summarized with random effects models. Intervention effects for smoking cessation were significant at posttreatment and comparable for participants in addictions treatment and recovery; however, intervention effects for smoking cessation were nonsignificant at long-term follow-up. Smoking cessation interventions provided during addictions treatment were associated with a 25% increased likelihood of long-term abstinence from alcohol and illicit drugs. Short-term smoking cessation effects look promising, but innovative strategies are needed for long-term cessation. Contrary to previous concerns, smoking cessation interventions during addictions treatment appeared to enhance rather than compromise long-term sobriety.

20 March 2017 in Annual Review of Public Health
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Journal Article
Open access
Judith J. ProchaskaJudith J. Prochaska
Judith J. Prochaska
SDSmita Das
Smita Das
Kelly C. Young-WolffKelly C. Young-Wolff
Kelly C. Young-Wolff
Tobacco use remains the leading preventable cause of death worldwide. In particular, people with mental illness are disproportionately affected with high smoking prevalence; they account for more than 200,000 of the 520,000 tobacco-attributable deaths in the United States annually and die on average 25 years prematurely. Our review aims to provide an update on smoking in the mentally ill. We review the determinants of tobacco use among smokers with mental illness, presented with regard to the public health HAVE framework of “the host” (e.g., tobacco user characteristics), the “agent” (e.g., nicotine product characteristics), the “vector” (e.g., tobacco industry), and the “environment” (e.g., smoking policies). Furthermore, we identify the significant health harms incurred and opportunities for prevention and intervention within a health care systems and larger health policy perspective. A comprehensive effort is warranted to achieve equity toward the 2025 Healthy People goal of reducing US adult tobacco use to 12%, with attention to all subgroups, including smokers with mental illness.

7 January 2014 in Circulation
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EMEdward J. Mills
Edward J. Mills
KTKristian Thorlund
Kristian Thorlund
SEShawn Eapen
Shawn Eapen
PWPing Wu
Ping Wu
Judith J. ProchaskaJudith J. Prochaska
Judith J. Prochaska
Background—: Stopping smoking is associated with many important improvements in health and quality of life. The use of cessation medications is recommended to increase the likelihood of quitting. However, there is historical and renewed concern that smoking cessation therapies may increase the risk of cardiovascular disease events associated within the quitting period. We aimed to examine whether the 3 licensed smoking cessation therapies—nicotine replacement therapy, bupropion, and varenicline—were associated with an increased risk of cardiovascular disease events using a network meta-analysis. Methods and Results—: We searched 10 electronic databases, were in communication with authors of published randomized, clinical trials (RCTs), and accessed internal US Food and Drug Administration reports. We included any RCT of the 3 treatments that reported cardiovascular disease outcomes. Among 63 eligible RCTs involving 21 nicotine replacement therapy RCTs, 28 bupropion RCTs, and 18 varenicline RCTs, we found no increase in the risk of all cardiovascular disease events with bupropion (relative risk [RR], 0.98; 95% confidence interval [CI], 0.54–1.73) or varenicline (RR, 1.30; 95% CI, 0.79–2.23). There was an elevated risk associated with nicotine replacement therapy that was driven predominantly by less serious events (RR, 2.29; 95% CI, 1.39–3.82). When we examined major adverse cardiovascular events, we found a protective effect with bupropion (RR, 0.45; 95% CI, 0.21–0.85) and no clear evidence of harm with varenicline (RR, 1.34; 95% CI, 0.66–2.66) or nicotine replacement therapy (RR, 1.95; 95% CI, 0.26–4.30). Conclusion—: Smoking cessation therapies do not appear to raise the risk of serious cardiovascular disease events.

1 April 2003 in American Journal of Preventive Medicine
Verified
Journal Article
Closed access
JSJames Sallis
James Sallis
TMThomas L McKenzie
Thomas L McKenzie
TCTerry L Conway
Terry L Conway
JEJohn P Elder
John P Elder
Judith J. ProchaskaJudith J. Prochaska
Judith J. Prochaska
JAJohn E Alcaraz
John E Alcaraz