Qualitative and quantitative changes in dogs’ information-seeking behaviours during the subsequent phases of operant conditioning training using a scent lineup, were investigated. Particular interest was paid to behaviours which may have an impact on errors committed by dogs at work in a scent lineup and thus on the reliability of the canine identification of humans on the base of scent. Significant individual differences were found in dogs’ performance in operant conditioning during match-to-sample trials. The style and time of sniffing the pattern scent sample as well as the searching time and number of stations sniffed in the lineup were found to influence the percentage of errors (false positive and false negative indications) made by dogs. The effects of the dogs’ routine e.g. omitting stations, using other cues than olfactory ones (visual) and effects of the non-verbal communications between handler and dog have been discussed.

The aim of the experiment was to test the effect of an elevated level of glucocorticoids on the mouse hippocampal transcriptome after 12 h of treatment with corticosterone that was administered during an active phase of the circadian cycle. Additionally, we also tested the circadian changes in gene expression and the decay time of transcriptomic response to corticosterone. Gene expression was analyzed using microarrays. Obtained results show that transcriptomic responses to glucocorticoids are heterogeneous in terms of the decay time with some genes displaying persistent changes in expression during 9 h of rest. We have also found a considerable overlap between genes regulated by corticosterone and genes implicated previously in stress response. The examples of such genes are Acer2, Agt, Apod, Aqp4, Etnppl, Fabp7, Fam107a, Fjx1, Fmo2, Galnt15, Gjc2, Heph, Hes5, Htra1, Jdp2, Kif5a, Lfng, Lrg1, Mgp, Mt1, Pglyrp1, Pla2g3, Plin4, Pllp, Ptgds, Ptn, Slc2a1, Slco1c1, Sult1a1, Thbd and Txnip. This indicates that the applied model is a useful tool for the investigation of mechanisms underlying the stress response.