Terry Huang

Prof. Dr. Terry Huang

Professor and Chair, Department of Health Policy and Management Director, Center for Systems and Community Design
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orcid

Research Focus & Metrics

Academic Career Length

27Years (2000-Present)

(Calculated once Scilit publication data is synced: current year − year of first publication + 1)

Representative Publications

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4 March 2020 in Nature Medicine
Verified
Journal Article
Open access
PDF file
FRFrancesco Rubino
Francesco Rubino
RPRebecca M. Puhl
Rebecca M. Puhl
DCDavid E. Cummings
David E. Cummings
RERobert H. Eckel
Robert H. Eckel
DRDonna H Ryan
Donna H Ryan
JDJohn B. Dixon
John B. Dixon
People with obesity commonly face a pervasive, resilient form of social stigma. They are often subject to discrimination in the workplace as well as in educational and healthcare settings. Research indicates that weight stigma can cause physical and psychological harm, and that affected individuals are less likely to receive adequate care. For these reasons, weight stigma damages health, undermines human and social rights, and is unacceptable in modern societies. To inform healthcare professionals, policymakers, and the public about this issue, a multidisciplinary group of international experts, including representatives of scientific organizations, reviewed available evidence on the causes and harms of weight stigma and, using a modified Delphi process, developed a joint consensus statement with recommendations to eliminate weight bias. Academic institutions, professional organizations, media, public-health authorities, and governments should encourage education about weight stigma to facilitate a new public narrative about obesity, coherent with modern scientific knowledge.

1 June 2015 in The Lancet
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Journal Article
Closed access
CRChristina A Roberto
Christina A Roberto
BSBoyd Swinburn
Boyd Swinburn
CHCorinna Hawkes
Corinna Hawkes
Terry HuangTerry Huang
Terry Huang
SCSergio A Costa
Sergio A Costa
KBKelly D Brownell
Kelly D Brownell

1 August 2011 in The Lancet
Verified
Journal Article
Closed access
SGSteven L Gortmaker
Steven L Gortmaker
BSBoyd A Swinburn
Boyd A Swinburn
DLDavid Levy
David Levy
RCRob Carter
Rob Carter
PMPatricia L Mabry
Patricia L Mabry
MMMarjory L Moodie
Marjory L Moodie

27 October 2021 in Nature Reviews Gastroenterology & Hepatology
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Journal Article
Closed access
PDF file
JLJeffrey Lazarus
Jeffrey Lazarus
HMHenry E. Mark
Henry E. Mark
QAQuentin M. Anstee
Quentin M. Anstee
JAJuan Pablo Arab
Juan Pablo Arab
RBRachel L. Batterham
Rachel L. Batterham
SZShira Zelber-Sagi
Shira Zelber-Sagi

27 September 2018 in Preventing Chronic Disease
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Journal Article
Closed access
MAMyra Altman
Myra Altman
Terry HuangTerry Huang
Terry Huang
JBJessica Y. Breland
Jessica Y. Breland
Preventing Chronic Disease (PCD) is a peer-reviewed electronic journal established by the National Center for Chronic Disease Prevention and Health Promotion. PCD provides an open exchange of information and knowledge among researchers, practitioners, policy makers, and others who strive to improve the health of the public through chronic disease prevention.

1 January 2004 in The Journal of Clinical Endocrinology & Metabolism
Verified
Journal Article
Open access
MCMartha L. Cruz
Martha L. Cruz
MWMarc J. Weigensberg
Marc J. Weigensberg
Terry HuangTerry Huang
Terry Huang
GBGeoff Ball
Geoff Ball
GSGabriel Q. Shaibi
Gabriel Q. Shaibi
MGMichael I. Goran
Michael I. Goran
The prevalence of the metabolic syndrome is highest among Hispanic adults. However, studies exploring the metabolic syndrome in overweight Hispanic youth are lacking. Subjects were 126 overweight children (8–13 yr of age) with a family history for type 2 diabetes. The metabolic syndrome was defined as having at least three of the following: abdominal obesity, low high-density lipoprotein (HDL) cholesterol, hypertriglyceridemia, hypertension, and/or impaired glucose tolerance. Insulin sensitivity was determined by the frequently sampled iv glucose tolerance test and minimal modeling. The prevalence of abdominal obesity, low HDL cholesterol, hypertriglyceridemia, systolic and diastolic hypertension, and impaired glucose tolerance was 62, 67, 26, 22, 4, and 27%, respectively. The presence of zero, one, two, or three or more features of the metabolic syndrome was 9, 22, 38, and 30%, respectively. After controlling for body composition, insulin sensitivity was positively related to HDL cholesterol (P < 0.01) and negatively related to triglycerides (P < 0.001) and systolic (P < 0.01) and diastolic blood pressure (P < 0.05). Insulin sensitivity significantly decreased (P < 0.001) as the number of features of the metabolic syndrome increased. In conclusion, overweight Hispanic youth with a family history for type 2 diabetes are at increased risk for cardiovascular disease and type 2 diabetes, and this appears to be due to decreased insulin sensitivity. Improving insulin resistance may be crucial for the prevention of chronic disease in this at-risk population.