Purpose: To analyze the effect of inspiratory muscle training (IMT) on the maximal inspiratory mouth pressure (MIP) and performance of elite swimmers. Methods: Eight participants performed a 3000-m swimming test (T-3000), followed by blood lactate measurements at 1 and 5 minutes postexercise.. The testing protocol was carried out before and after 6 weeks of IMT, in which a high-volume IMT group (HV-IMT) (n = 4) performed IMT twice a day—in the morning in a seated position and in the afternoon in a concurrent session of IMT and core muscle training. Also, a low-volume IMT group (LV-IMT) (n = 4) performed IMT in the morning session only. Results: After the intervention, both groups improved their MIP, HV-IMT (132.75 [27.42] to 156.75 [21.88] cmH₂O; P = .010; d = 0.967) and LV-IMT (149.25 [22.82] to 171.50 [23.74] cmH₂O; P = .013; d = 0.955), without a significant difference between groups (P = .855). Regarding swimming performance, there were no changes between groups in the T-3000 (P = .472) or lactate removal rate (P = .104). Conclusion: IMT increased inspiratory muscle strength in elite swimmers, but there was no association or meaningful impact on swimming performance.

Purpose: Intermittent pneumatic compression (IPC) applies gradual pressure to facilitate lymph and blood flow movement to reduce exercise-induced tissue fluid accumulation and plasma volume loss. This study aimed to evaluate the cardiovascular system response during the recovery with IPC compared with passive recovery (Sham). Methods: Sixteen volunteers (7 females and 9 males) executed a cycling-based exhausting sprint interval exercise (8 × 20 s all out), followed by a 30-min IPC or Sham condition. Participants performed two trials in a randomised, counterbalanced, and crossover design. Several cardiovascular parameters (blood pressure, heart function, and peripheral vascular resistance) were recorded at baseline (5ʹ), through the recovery protocol (30ʹ), and afterwards (5ʹ). Results: The use of IPC during the recovery phase led to a faster recovery, stated in relative values to pre-exercise, in mean blood pressure (102.5 ± 19.3% vs. 92.7 ± 12.5%; P < 0.001), and cardiac output (139.8 ± 30.0% vs. 146.2 ± 40.2%; P < 0.05) in comparison to Sham condition. Furthermore, during the IPC-based recovery, there was a slower recovery in cardiac pressure change over time (92.5 ± 25.8% vs. 100.5 ± 48.9%; P < 0.05), and a faster return to pre-exercise values in the peripheral vascular resistance (75.2 ± 25.5% vs. 64.8 ± 17.4%; P < 0.001) compared to Sham. Conclusion: The application of IPC after high-intensity exercise promotes the recovery of the cardiovascular system, reducing cardiovascular strain. Future investigations should consider the effects on the sympathetic-parasympathetic balance, such as heart rate variability, to assess further bonds between the use of IPC and autonomous control.

The aim of the present research was to examine the stroking rate (SR) values of successful and non-successful swimmers in the 10 km and 25 km races of the FINA 2019 World Swimming Championships. Data from 175 participants (95 men and 80 female) were classified according to their finishing positions. There were no meaningful differences in the overall SR values displayed by successful or non-successful participants during the 10 km and 25 km open water races of the FINA 2019 World Swimming Championships. However, there were changes in the SR throughout the races that depended on the swimmer’s performance group and gender. Successful swimmers in the 10 km event typically displayed even SR in the first 5 km but, unlike the remaining performance groups, increased their SR at some point in the second 5 km of the race. In the 25 km race, successful female swimmers presented an even SR profile for most of the race, whereas successful males presented a more variable profile. Nevertheless, no relationships between partial or average SR and finishing positions occurred, either in the 10 km or in the 25 km race. Changes in the SR values should be included in the race plan of open water swimmers according to tactical and pacing strategies.

Artistic swimmers (AS) are exposed to repeated apnoeas in the aquatic environment during high intensity exercise provoking specific physiological responses to training, apnoea, and immersion. This study aimed to evaluate the changes in lung diffusing capacity in AS pre-, mid- and post-training in a combined session of apnoeic swimming, figures and choreography. Eleven elite female AS from the Spanish national team were the study’s participants. The single-breath method was used to measure lung diffusing capacity for carbon monoxide (DL_CO) and one-way repeated measures ANOVA was utilized to evaluate the statistical analysis. Basal values of DL_CO were higher than normal for their age and height (33.6±4.9 mL·min⁻¹·mmHg⁻¹; 139±19%) and there were a significant interaction between DL_CO and AS training (ŋ² _p=0.547). After the apnoeic swimming (mid-training) there was an increase in DL_CO from basal to 36.7±7.3 mL·min⁻¹·mmHg⁻¹ (p=0.021), and after the figures and choreography (post-training) there was a decrease compared to mid-training (32.3±4.6 mL·min⁻¹·mmHg⁻¹, p=0.013). Lung diffusing capacity changes occur during AS training, including a large increase after apnoeic swimming. There were no differences in lung diffusing capacity from pre- to post-training, although large inter-individual variability was observed. Received: 15 May 2020 Accepted: 19 June 2020 Publication Date: 26 August 2020 (online) © 2020. Thieme. All rights reserved. © Georg Thieme Verlag KG Stuttgart · New York

Swimming exercise at sea level causes a transient decrease in lung diffusing capacity for carbon monoxide (DL_CO). The exposure to hypobaric hypoxia can affect lung gas exchange, and hypoxic pulmonary vasoconstriction may elicit pulmonary oedema. The purpose of this study is to evaluate whether there are changes in DL_CO during a 14-day altitude training camp (1850 m) in elite swimmers and the acute effects of a combined training session of swimming in moderate hypoxia and 44-min cycling in acute normobaric severe hypoxia (3000 m). Participants were eight international level swimmers (5 females and 3 males; 17–24 years old; 173.5 ± 5.5 cm; 64.4 ± 5.3 kg) with a training volume of 80 km per week. The single-breath method was used to measure the changes in DL_CO and functional gas exchange parameters. No changes in DL_CO after a 14-day altitude training camp at 1850 m were detected but a decrease in alveolar volume (VA; 7.13 ± 1.61 vs. 6.50 ± 1.59 L; p = 0.005; d = 0.396) and an increase in the transfer coefficient of the lung for carbon monoxide (K_CO; 6.23 ± 1.03 vs. 6.83 ± 1.31 mL·min⁻¹·mmHg⁻¹·L⁻¹; p = 0.038; d = 0.509) after the altitude camp were observed. During the acute hypoxia combined session, there were no changes in DL_CO after swimming training at 1850 m, but there was a decrease in DL_CO after cycling at a simulated altitude of 3000 m (40.6 ± 10.8 vs. 36.8 ± 11.2 mL·min⁻¹·mmHg⁻¹; p = 0.044; d = 0.341). A training camp at moderate altitude did not alter pulmonary diffusing capacity in elite swimmers, although a cycling session at a higher simulated altitude caused a certain degree of impairment of the alveolar–capillary gas exchange.