Dr. Wen-Xing Ding became an assistant professor in 2009 and then a professor at the Department of Pharmacology, Toxicology, and Therapeutics in 2017 at The University of Kansas Medical Center. He graduated from Shanghai Medical University (now Fudan University) in China in 1992 in Preventive Medicine, and then obtained a Master's degree in Toxicology in 1995 at Shanghai Medical University. He then got his PhD in Molecular Toxicology in 2001 from the National University of Singapore. He then did his Postdoc training at the University of Pittsburgh in 2001 and became a research assistant professor at the Department of Pathology in 2006 at the University of Pittsburgh. He has been working on the role of autophagy in alcohol and drug-induced liver injury since 2009. He is particularly interested in how autophagy selectively removes cellular damaged/excess organelles such as mitochondria and lipid droplets in hepatocytes.
Research Keywords & Expertise
Autophagy
Lipid Metabolism
mitophagy
ER stress
liver injury
Alcohol and non-alcoho...
Protein aggregates
Fingerprints
67%
Autophagy
47%
liver injury
23%
mitophagy
5%
Lipid Metabolism
5%
Protein aggregates
5%
ER stress
5%
Alcohol and non-alcoholic liver diseases
Short Biography
Dr. Wen-Xing Ding became an assistant professor in 2009 and then a professor at the Department of Pharmacology, Toxicology, and Therapeutics in 2017 at The University of Kansas Medical Center. He graduated from Shanghai Medical University (now Fudan University) in China in 1992 in Preventive Medicine, and then obtained a Master's degree in Toxicology in 1995 at Shanghai Medical University. He then got his PhD in Molecular Toxicology in 2001 from the National University of Singapore. He then did his Postdoc training at the University of Pittsburgh in 2001 and became a research assistant professor at the Department of Pathology in 2006 at the University of Pittsburgh. He has been working on the role of autophagy in alcohol and drug-induced liver injury since 2009. He is particularly interested in how autophagy selectively removes cellular damaged/excess organelles such as mitochondria and lipid droplets in hepatocytes.