Peter Hassmén

Dr. Peter Hassmén


Research Focus & Metrics

Academic Career Length

40Years (1987-Present)

(Calculated once Scilit publication data is synced: current year − year of first publication + 1)

Representative Publications

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1 July 1998 in Sports Medicine
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Journal Article
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GKGöran Kenttä
Göran Kenttä
PHPeter Hassmén
Peter Hassmén

1 September 1987 in European Journal of Applied Physiology
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Journal Article
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GBGunnar Borg
Gunnar Borg
PHPeter Hassmén
Peter Hassmén
MLMonica Lagerström
Monica Lagerström

1 January 2000 in Preventive Medicine
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Journal Article
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PHPeter Hassmén
Peter Hassmén
NKNathalie Koivula
Nathalie Koivula
AUAntti Uutela
Antti Uutela

15 March 2017 in Clinical Obesity
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Journal Article
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EBE. Burgess
E. Burgess
PHPeter Hassmén
Peter Hassmén
KPKate Pumpa
Kate Pumpa

1 May 1996 in The Journal of the Acoustical Society of America
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Journal Article
Closed access
BBBirgitta Berglund
Birgitta Berglund
PHPeter Hassmén
Peter Hassmén
RJR. F. Soames Job
R. F. Soames Job
The sources of human exposure to low-frequency noise and its effects are reviewed. Low-frequency noise is common as background noise in urban environments, and as an emission from many artificial sources: road vehicles, aircraft, industrial machinery, artillery and mining explosions, and air movement machinery including wind turbines, compressors, and ventilation or air-conditioning units. The effects of low-frequency noise are of particular concern because of its pervasiveness due to numerous sources, efficient propagation, and reduced efficacy of many structures (dwellings, walls, and hearing protection) in attenuating low-frequency noise compared with other noise. Intense low-frequency noise appears to produce clear symptoms including respiratory impairment and aural pain. Although the effects of lower intensities of low-frequency noise are difficult to establish for methodological reasons, evidence suggests that a number of adverse effects of noise in general arise from exposure to low-frequency noise: Loudness judgments and annoyance reactions are sometimes reported to be greater for low-frequency noise than other noises for equal sound-pressure level; annoyance is exacerbated by rattle or vibration induced by low-frequency noise; speech intelligibility may be reduced more by low-frequency noise than other noises except those in the frequency range of speech itself, because of the upward spread of masking. On the other hand, it is also possible that low-frequency noise provides some protection against the effects of simultaneous higher frequency noise on hearing. Research needs and policy decisions, based on what is currently known, are considered.

1 December 1994 in International Journal of Behavioral Medicine
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Journal Article
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ULUlf Lundberg
Ulf Lundberg
RKRoland Kadefors
Roland Kadefors
BMBo Melin
Bo Melin
GPGunnar Palmerud
Gunnar Palmerud
PHPeter Hassmén
Peter Hassmén
IDIngela Elfsberg Dohns
Ingela Elfsberg Dohns
Although it is generally assumed that mental stress induces muscular tension, the experimental data have, so far, been inconclusive. Likely explanations for these inconsistent findings are (a) too small subject samples in some experiments, (b) the use of only one type of stress stimulation, and (c) the lack of objective (physiological) measurements documenting the stress-inducing properties of the experimental treatment. Furthermore, the effect of mental stress and physical load separately, versus the combined influence of physical and mental load on muscular tension, has not been investigated earlier. Therefore. the aim of the present experiment was to examine the effects of mental stress as well as of physical load, separately and in combination, on perceived stress, physiological stress responses, and on muscular tension as reflected in electromyographical (EMG) activity of the trapezius muscle. Sixty two female subjects were individually exposed to mental arithmetic, the Stroop color word test (CWT), the cold pressor test, standardized test contractions (TCs), and the CWT combined with a TC. Compared to baseline, the stress session induced significant increases in systolic and diastolic blood pressure, heart rate, urinary catecholamines, salivary Cortisol, and self-reported stress. Each of the two mental stress tests induced a significant increase in EMG activity, The CWT caused a rise in EMG activity also during the TC, which was significantly more pronounced than the increase induced by the CWT alone. Blood pressure responses and self-reported stress followed the same pattern as the EMG activity. The results are consistent with the assumption that psychological stress plays a role in musculoskeletal disorders by increasing muscular tension both in low-load work situations and in the absence of physical load. It is also indicated that the stress-induced increase in muscular tension is accentuated on top of a physical load.