Katarzyna Leźnicka

Prof. Katarzyna Leźnicka

orcid

Research Focus & Metrics

Academic Career Length

12Years (2015-Present)

(Calculated once Scilit publication data is synced: current year − year of first publication + 1)

Representative Publications

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1 December 2015 in Journal of Human Kinetics
Verified
Journal Article
Open access
MCMonika Chudecka
Monika Chudecka
Anna LubkowskaAnna Lubkowska
Anna Lubkowska
KLKatarzyna Leźnicka
Katarzyna Leźnicka
KKKrzysztof Krupecki
Krzysztof Krupecki
In order to achieve higher efficiency of training and thus better athletic performance, new research and diagnostic methods are constantly being developed, particularly those that are non-invasive. One such a method is thermography, suitable for quantitative and therefore objective evaluation of variables, such as changes in the temperature of the skin covering working muscles. The aim of this study was to use a thermal imaging infrared camera to evaluate temperature changes of symmetric body surfaces over symmetrically working muscles of male scullers after exercising on a two-oared rowing ergometer and compare these to asymmetrically working muscles of handball players after an endurance training session containing elements of an actual game. In the scullers, the mean temperature of body surfaces was always lower post than pre exercise, with no significant differences in an average temperature drop between the opposite sides, indicating that the work of the muscles involved in the physical exertion on the rowing ergometer was symmetrical. In contrast, in the handball players, skin temperatures in symmetric areas over the asymmetrically working muscles showed statistically significant differences between sides, which was associated with the functional asymmetry of training. This study indicates that thermal imaging may be useful for coaches in the evaluation of technical preparations in sports in which equal involvement of symmetric muscles is a condition of success, e.g. in scullers.

31 August 2020 in Journal of Human Kinetics
Verified
Journal Article
Open access
AMAgnieszka Maciejewska-Skrendo
Agnieszka Maciejewska-Skrendo
KLKatarzyna Leźnicka
Katarzyna Leźnicka
ALAgata Leońska-Duniec
Agata Leońska-Duniec
MWMichal Wilk
Michal Wilk
Aleksandra Filip-StachnikAleksandra Filip-Stachnik
Aleksandra Filip-Stachnik
MSMarek Sawczuk
Marek Sawczuk
Muscle stiffness, muscle elasticity and explosive strength are the main components of athletes’ performance and they show a sex-based as well as ethnicity variation. Muscle stiffness is thought to be one of the risk factors associated with sports injuries and is less common in females than in males. These observations may be explained by circulating levels of sex hormones and their specific receptors. It has been shown that higher levels of estrogen are associated with lower muscle stiffness responsible for suppression of collagen synthesis. It is thought that these properties, at least in part, depend on genetic factors. Particularly, the gene encoding estrogen receptor 1 (ESR1) is one of the candidates that may be associated with muscle stiffness. Muscle elasticity increases with aging and there is evidence suggesting that titin (encoded by the TTN gene), a protein that is expressed in cardiac and skeletal muscles, is one of the factors responsible for elastic properties of the muscles. Mutations in the TTN gene result in some types of muscular dystrophy or cardiomyopathy. In this context, TTN may be regarded as a promising candidate for studying the elastic properties of muscles in athletes. The physiological background of explosive strength depends not only on the muscle architecture and muscle fiber composition, but also on the central nervous system and functionality of neuromuscular units. These properties are, at least partly, genetically determined. In this context, the ACTN3 gene code for α-actinin 3 has been widely researched.

1 October 2017 in Physiology & Behavior
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Journal Article
Closed access
KLKatarzyna Leźnicka
Katarzyna Leźnicka
ASAnna Starkowska
Anna Starkowska
MTMaciej Tomczak
Maciej Tomczak
Pawel CieszczykPawel Cieszczyk
Pawel Cieszczyk
MBMonika Białecka
Monika Białecka
MPMaciej Pawlak
Maciej Pawlak

1 January 2019 in Biology of Sport
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Journal Article
Open access
PDF file
ELEwelina Lulińska-Kuklik
Ewelina Lulińska-Kuklik
KLKatarzyna Leźnicka
Katarzyna Leźnicka
KHKinga Humińska-Lisowska
Kinga Humińska-Lisowska
WMWaldemar Moska
Waldemar Moska
MMMonika Helena Michałowska-Sawczyn
Monika Helena Michałowska-Sawczyn
ALAgata Leońska-Duniec
Agata Leońska-Duniec
The aim of the present study was to analyse VEGFA rs699947, rs1570360, and rs2010963 polymorphisms with susceptibility to anterior cruciate ligament rupture (ACLR) in a Polish population. The study included 412 physically active Caucasian participants. The study group consisted of 222 individuals...

18 December 2022 in Nutrients
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Journal Article
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AMAgnieszka Maciejewska-Skrendo
Agnieszka Maciejewska-Skrendo
MMMyosotis Massidda
Myosotis Massidda
FTFilippo Maria Tocco
Filippo Maria Tocco
KLKatarzyna Leźnicka
Katarzyna Leźnicka
Genetic components may play an important role in the regulation of nutrient and energy metabolism. In the presence of specific genetic variants, metabolic dysregulation may occur, especially in relation to the processes of digestion, assimilation, and the physiological utilization of nutrients supplied to the body, as well as the regulation of various metabolic pathways and the balance of metabolic changes, which may consequently affect the effectiveness of applied reduction diets and weight loss after training. There are many well-documented studies showing that the presence of certain polymorphic variants in some genes can be associated with specific changes in nutrient and energy metabolism, and consequently, with more or less desirable effects of applied caloric reduction and/or exercise intervention. This systematic review focused on the role of genes encoding peroxisome proliferator-activated receptors (PPARs) and their coactivators in nutrient and energy metabolism. The literature review prepared showed that there is a link between the presence of specific alleles described at different polymorphic points in PPAR genes and various human body characteristics that are crucial for the efficacy of nutritional and/or exercise interventions. Genetic analysis can be a valuable element that complements the work of a dietitian or trainer, allowing for the planning of a personalized diet or training that makes the best use of the innate metabolic characteristics of the person who is the subject of their interventions.

1 March 2017 in Physiological Genomics
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Journal Article
Closed access
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AZAleksandra Zarebska
Aleksandra Zarebska
Zbigniew JastrzębskiZbigniew Jastrzębski
Zbigniew Jastrzębski
IAIldus I. Ahmetov
Ildus I. Ahmetov
PZPiotr Zmijewski
Piotr Zmijewski
Pawel CieszczykPawel Cieszczyk
Pawel Cieszczyk
AMAgnieszka Maciejewska-Skrendo
Agnieszka Maciejewska-Skrendo
The GSTP1 gene encodes glutathione S-transferase P1, which is a member of the glutathione S-transferases (GSTs), a family of enzymes playing an important role in detoxification and in the antioxidant defense system. There is some evidence indicating that GSTP1 c.313A>G polymorphism may be beneficial for exercise performance. Therefore, we decided to verify the association between the frequency of GSTP1 c.313A>G variants, physical performance, and athletes' status in two cohorts: in a group of Russian athletes ( n = 507) and in an independent population of Polish athletes ( n = 510) in a replication study. The initial association study conducted with the Russian athletes revealed that the frequency of the minor G allele was significantly higher in all athletes than in controls; that was confirmed in the replication study of Polish athletes. In the combined cohort, the differences between athletes ( n = 1017) and controls ( n = 1246) were even more pronounced (32.7 vs 25.0%, P < 0.0001). Our findings emphasize that the G allele of the GSTP1 gene c.313A>G single nucleotide polymorphism is associated with improved endurance performance. These observations could support the hypothesis that the GSTP1 G allele may improve exercise performance by better elimination of exercise-induced ROS.