The identification of hazardous chemicals based on hyperspectral imaging is an important emergent means for the prevention of explosion accidents and the early warning of secondary hazards. In this study, we used a combination of spectral curve matching based on full-waveform characteristics and spectral matching based on spectral characteristics to identify the hazardous chemicals, and proposed a method to quantitatively characterize the matching degree of the spectral curves of hazardous chemicals. The results showed that the four hazardous chemicals, sulfur, red phosphorus, potassium permanganate, and corn starch had bright colors, distinct spectral curve characteristics, and obvious changes in reflectivity, which were easy to identify. Moreover, the matching degree of their spectral curves was positively correlated with their reflectivity. However, the spectral characteristics of carbon powder, strontium nitrate, wheat starch, and magnesium–aluminum alloy powder were not obvious, with no obvious characteristic peaks or trends of change in reflectivity. Except for the reflectivity and the matching degree of the carbon powder being maintained at a low level, the reflectivity of the remaining three samples was relatively close, so that it was difficult to identify with the spectral curves alone, and color information should be considered for further identification.

The purpose of this study was to explore the damage effects and injury mechanism of free‐field explosion shock waves on rabbit lungs. Six free‐field explosion experiments, each with 500 g trinitrotoluene (TNT), were conducted as the shock wave overpressure acting on the rabbits was measured. The peak overpressure of the shock wave was 533, 390, 249, 102, and 69 kPa at the respective test points. Damage to the rabbit lungs caused by shock wave overpressure was investigated through observation, anatomical analysis, and hematoxylin‐eosin (HE) staining processing. The shock wave overpressure of 69–102 kPa caused mild‐to‐moderate injury; the shock wave overpressure of 102–249 kPa caused moderate injury; the shock wave overpressure of 249–390 kPa resulted in moderate‐to‐severe injury; and the shock wave overpressure of 390–533 kPa caused severe injury to the rabbit. Mild, moderate, and severe injuries destroyed some, most, or all alveolar structures, correspondingly, as well as producing partial cell apoptosis. The overpressure damage mechanism primarily involves the collapse and rupture of pulmonary alveolus in the lung tissue. As a novel attempt, the investigation provided here may serve to improve the current shock wave injury mechanism.