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Liang-Jun Yan

Prof. Liang-Jun Yan

College of Pharmacy, University of North Texas Health Science Center, Fort Worth...

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Dr. Liang-Jun Yan is a Professor of Pharmaceutical Sciences and a member of the Institute for Healthy Aging at the University of North Texas Health Science Center. He received his bachelor's degree in Biochemistry from Peking University and completed his master's studies in Biochemistry at the Institute of Biophysics Chinese Academy of Sciences and his PhD studies in Molecular and Cell Biology at the University of California Berkeley. His research focuses on oxidative stress, age-related metabolic disorders, diabetic kidney disease, and acute kidney injury. His lab’s current projects focus on kidney diseases encompassing ischemia- and drug-induced acute kidney injury and diabetic kidney disease. These projects aim to understand the molecular basis of oxidative stress and redox dysregulation in these kidney diseases and identify novel targets for developing potential therapeutic approaches.

Research Keywords & Expertise

Diabetes
Oxidative Stress
mitochondrial dysfunct...
neuroprotection
protein oxidation

Fingerprints

34%
Oxidative Stress
26%
Diabetes
17%
neuroprotection
15%
protein oxidation
9%
Redox imbalance
5%
mitochondrial dysfunction

Short Biography

Dr. Liang-Jun Yan is a Professor of Pharmaceutical Sciences and a member of the Institute for Healthy Aging at the University of North Texas Health Science Center. He received his bachelor's degree in Biochemistry from Peking University and completed his master's studies in Biochemistry at the Institute of Biophysics Chinese Academy of Sciences and his PhD studies in Molecular and Cell Biology at the University of California Berkeley. His research focuses on oxidative stress, age-related metabolic disorders, diabetic kidney disease, and acute kidney injury. His lab’s current projects focus on kidney diseases encompassing ischemia- and drug-induced acute kidney injury and diabetic kidney disease. These projects aim to understand the molecular basis of oxidative stress and redox dysregulation in these kidney diseases and identify novel targets for developing potential therapeutic approaches.