Resat Cinar obtained a B.Sc. in Pharmacy degree from Ege University, the Faculty of Pharmacy, Izmir, Turkey, in 2004. He obtained an M.Sc. in Neurobiology and Physiology in 2009, followed by a Ph.D. in Biological Sciences in 2010, from the University of Szeged, Hungary. He also earned an M.B.A. degree from the Faculty of Economics and Business Administration, at Szeged University. He was a research associate between 2005 and 2009 at the Institute of Biochemistry at the Hungarian Academy of Sciences Biological Research Center in Szeged, Hungary, where he studied the molecular pharmacology of cannabinoid receptor 1 (CB1R) and opioid receptors. In 2015, he became a staff scientist at the Laboratory of Physiologic Studies in the NIAAA. In 2021, he was recruited as a tenure-track investigator and acting chief of the Section on Fibrotic Disorders. He was one of the recipients of the NIH NCATS Director’s Award for Scientific and Medical Achievement in 2018. His research demonstrated the pathologic roles of the endocannabinoid/CB1R system in fibroproliferative disorders such as lung and liver fibrosis.
Resat Cinar obtained a B.Sc. in Pharmacy degree from Ege University, the Faculty of Pharmacy, Izmir, Turkey, in 2004. He obtained an M.Sc. in Neurobiology and Physiology in 2009, followed by a Ph.D. in Biological Sciences in 2010, from the University of Szeged, Hungary. He also earned an M.B.A. degree from the Faculty of Economics and Business Administration, at Szeged University. He was a research associate between 2005 and 2009 at the Institute of Biochemistry at the Hungarian Academy of Sciences Biological Research Center in Szeged, Hungary, where he studied the molecular pharmacology of cannabinoid receptor 1 (CB1R) and opioid receptors. In 2015, he became a staff scientist at the Laboratory of Physiologic Studies in the NIAAA. In 2021, he was recruited as a tenure-track investigator and acting chief of the Section on Fibrotic Disorders. He was one of the recipients of the NIH NCATS Director’s Award for Scientific and Medical Achievement in 2018. His research demonstrated the pathologic roles of the endocannabinoid/CB1R system in fibroproliferative disorders such as lung and liver fibrosis.