Implantable loop recorders (ILRs) are vital for continuous rhythm monitoring; however, post-implantation migration can impair device function. ILR migration may range from minor positional shifts causing discomfort to severe displacement, potentially resulting in device malfunction or requiring surgical intervention. This review examines migration patterns to identify factors associated with ILR migration. A systematic literature search was conducted in PubMed, Cochrane Library, CINAHL, and EMBASE for case reports on ILR migration from inception to October 2024. Data on patient demographics, comorbidities, device models, implantation sites, detection times, and interventions were qualitatively synthesised to identify factors linked to migration. Older age, female gender, and specific comorbidities emerged as migration risk factors. Device implantation angulation and depth were common contributors. Migration typically followed a posterior or inferior direction and was detected within 5–35 days, often presenting as loss of connection or continuous chest pain. Migration was also observed following patient manipulation of the device. Although migration is rare, cases requiring video-assisted thoracoscopic surgery (VATS) highlight the significant morbidity associated with this complication. Optimising implantation techniques and employing effective follow-up strategies can reduce the risk of migration and improve migration detection. Further studies with standardised reporting are needed to better understand this complication.

The diagnosis of decompression sickness (DCS) is mostly based on clinical suspicion, and there is currently no available modality to fully confirm the diagnosis. However, the use of echocardiography in suspected DCS cases has become more common. In this case, transthoracic echocardiography (TTE) was used to detect microbubbles in the right cardiac chambers and monitor the patient after hyperbaric oxygen therapy (HBOT), suggesting the possible applicability of TTE in diagnosing and monitoring DCS patients. This report describes a 54‐year‐old Fisherman who was referred to the emergency department with dyspnea and mild confusion after a rapid ascent of a saturation dive at 50 m sea depth. After the initial evaluation, he was assessed using TTE to exclude the presence of structural heart disease, where it surprisingly showed spontaneous echo contrast inside the right cardiac chambers similar to agitated saline echo testing. The patient was then admitted for HBOT and follow‐up; rapid improvement was noticed after the first HBOT session and the TTE findings were fully resolved. TTE could be considered in the initial workup when DCS is suspected, and it might have a role in monitoring DCS patients if echocardiographic findings of bubble formation were documented in the pre‐hyperbaric therapy settings.

Background:Undeflatable coronary balloons during percutaneous coronary intervention (PCI) are rare but particularly hazardous because persistent intracoronary inflation can cause abrupt coronary occlusion, ischaemia, and haemodynamic collapse. We aimed to synthesise the published experience to define mechanistic patterns, identify recurring predisposing factors, and propose a practical bailout algorithm.Methods and Results:We performed a case-based review of PubMed, the Cochrane Library, and EBSCO from inception to February 2026. Eligible reports described balloons that could not be deflated while positioned in a coronary artery. A total of 15 coronary cases (2011–2025) met the inclusion criteria. Events occurred most often during post-dilatation or after stent deployment in complex anatomy, frequently involving severe calcification, aorto-ostial segments, chronic total occlusion-like lesions, and settings with limited guide engagement. Mechanistic patterns clustered into (i) intact-system non-deflation, where restoration of an exit pathway (system checks, sustained negative suction, and contrast dilution), followed by controlled decompression (wire puncture or mechanical- or energy-assisted disruption), was feasible; (ii) shaft-compromised events (fracture or detachment), where management shifted from decompression to mechanical retrieval, with surgery reserved for percutaneous failure.Conclusion:Undeflatable coronary balloons represent distinct failure modes with different viable rescue options. Outcomes appear to be driven by rapid restoration of perfusion and early recognition of shaft integrity. A structured escalation approach prioritising stabilisation, low-risk troubleshooting, controlled decompression, and decisive transition to retrieval, exclusion, or surgery when decompression is impossible could reduce secondary iatrogenic harm.

Tumor necrosis factor alpha inhibitors have transformed management of autoimmune diseases, but cardiovascular safety profile of these diseases remains an area of investigation. A 39-year-old woman with rheumatoid arthritis presented with an ST-segment elevation myocardial infarction 3 weeks after initiating adalimumab therapy. Coronary angiography revealed a long thrombus in the mid left anterior descending coronary artery with preserved TIMI flow grade 3 and no coronary stenosis. A multidisciplinary team elected to pursue conservative management with glycoprotein IIb/IIIa inhibition (tirofiban) without stent placement given the patient's young age, absence of atherosclerotic plaque, and normal right and circumflex coronary arteries. Repeat angiography after 72 hours demonstrated complete thrombus resolution. This case raises the possibility of a prothrombotic effect linked to adalimumab. Registry data suggest a potential increased risk of thrombotic cardiovascular events with this medication. Clinicians should maintain a high index of suspicion for arterial thrombosis in patients receiving adalimumab.

Background: While tailored information might have the potential to motivate stroke survivors to make essential lifestyle changes and improve long-term outcomes, how this varies among different stroke populations is not yet fully understood. Method: From November 2022 to May 2023, stroke survivors in the UK, who were clinically stable, participated in a community-based, descriptive cross-sectional study. Participants rated several information themes on a Likert scale from one to five, indicating the relevance of each information group to them. Data were analysed using Wilcoxon and chi-squared tests on SPSS. Descriptive statistics were employed for examining the preferred information delivery method, timing, personnel, and frequency. Results: Seventy survivors, with an average age of 67 ± 19 (61% males), were recruited. Survivors emphasised the importance of symptoms, risk factors, and recovery information during hospital stay, while medication and lifestyle change information were more significant in the community. Subgroup analysis revealed distinct patterns: First-time stroke survivors highlighted the importance of social and financial support (acute phase median Likert score 3, chronic phase median Likert score 4; p < 0.01), while those with prior strokes emphasised information on driving and working after stroke (acute phase median Likert score 4, chronic phase median Likert score 3; p < 0.05). Survivors recruited after six months of stroke prioritised knowledge of carer support in the community (acute phase median Likert score 3.5, chronic phase median Likert score 4; p < 0.01). Conclusion: Survivors’ information needs differ depending on factors such as the recovery phase, type of stroke, time since diagnosis, and the presence of a previous stroke. Considering these factors is essential when developing or providing information to stroke survivors.

Among the amazing turn of events in preventing and reducing the risk of cardiovascular diseases since the discovery of statins, thousand of physicians started to prescribe it as a regular life-long treatment, but regarding the expanding number of patients on statin therapy, a wide spectrum of side effects started to appear. Statin-associated myopathy considered as one of the most common side effects and could be subtle for a long time, we performed a review to provide a clinical summary of statin-associated myopathy and to discuss possible mechanisms of risk factors and management of statin-associated myopathy.