The family Geminiviridae includes plant-infecting viruses whose genomes are composed of one or two circular non-enveloped ssDNAs(+) of about 2.5–5.2 kb each in size. These insect-transmissible geminiviruses cause significant crop losses across continents and pose a serious threat to food security. Under the control of promoters generally located within the intergenic region, their genomes encode five to eight ORFs from overlapping viral transcripts. Most proteins encoded by geminiviruses perform multiple functions, such as suppressing defense responses, hijacking ubiquitin-proteasomal pathways, altering hormonal responses, manipulating cell cycle regulation, and exploiting protein-signaling cascades. Geminiviruses establish complex but coordinated interactions with several host elements to spread and facilitate successful infection cycles. Consequently, plants have evolved several multilayered defense strategies against geminivirus infection and distribution. Recent studies on the evasion of host-mediated resistance factors by various geminivirus proteins through novel mechanisms have provided new insights into the development of antiviral strategies against geminiviruses. This review summarizes the current knowledge concerning virus movement within and between cells, as well as the recent advances in our understanding of the biological roles of virus-encoded proteins in manipulating host-mediated responses and insect transmission. This review also highlights unexplored areas that may increase our understanding of the biology of geminiviruses and how to combat these important plant pathogens.

Whitefly (Bemisia tabaci)-transmitted begomoviruses cause severe diseases in numerous economically important dicotyledonous plants. Okra enation leaf curl disease (OELCuD) has emerged as a serious threat to okra (Abelmoschus esculentus L. Moench) cultivation in the Indian subcontinent. This study reports the association of a monopartite begomovirus (bhendi yellow vein mosaic virus; BYVMV) and betasatellite (bhendi yellow vein mosaic betasatellite; BYVB) with OELCuD in the Mau region of Uttar Pradesh, India. The BYVMV alone inoculated Nicotiana benthamiana and A. esculentus cv. Pusa Sawani plants developed mild symptoms. Co-inoculation of BYVMV and BYVB resulted in a reduced incubation period, an increased symptom severity, and an enhanced BYVMV accumulation by Southern hybridization and quantitative real-time PCR. This is the first study that satisfies Koch’s postulates for OELCuD in its natural host. Activities of various antioxidative enzymes were significantly increased in the virus-inoculated okra plants. Differential responses in various biochemical components (such as photosynthetic pigments, phenol, proline, and sugar) in diseased okra plants were observed. This change in phytochemical responses is significant in understanding its impact on virus pathogenesis and disease development.

Over the past several decades, xenobiotic chemicals have badly affected the environment including human health, ecosystem and environment. Animal-sourced biopolymers have been employed for the removal of heavy metals and organic dyes from the contaminated soil and waste waters. Animal-sourced biopolymers are biocompatible, cost-effective, eco-friendly, and sustainable in nature which make them a favorable choice for the mitigation of xenobiotic and hazardous compounds. Chitin/chitosan, collagen, gelatin, keratin, and silk fibroin-based biopolymers are the most commonly used biopolymers. This chapter reviews the current challenge faced in applying these animal-based biopolymers in eliminating/neutralizing various recalcitrant chemicals and dyes from the environment. This chapter ends with the discussion on the recent advancements and future development in the employability of these biopolymers in such environmental applications.

As plant-infecting viruses are intracellular obligate parasites, they rely heavily on the host (plant) machinery to complete every stage of its infection cycle including replication, transcription, pathogenesis and spread. Such plant-virus interactions include both host and nonhost interactions. Host interactions often cause successful infection and induce pathogenesis of the viruses within the host cells (host susceptibility) (Garcia-Ruiz, 2018). While the non-host interactions are non-compatible in nature that usually does not result in virus infection (host resistance) (Kang et al., 2005). In susceptible hosts, viruses do masterly redirect various host cellular machineries by interacting with various host factors to complete its infection cycle (Wang, 2015). Whereas in the resistant plants, host's natural resistance (defense) mechanisms restrict or limit the virus infection and disease development within the infection site (Baulcombe, 2004