Suzanne M Bertisch

Dr. Suzanne M Bertisch

orcid

Research Focus & Metrics

Academic Career Length

21Years (2006-Present)

(Calculated once Scilit publication data is synced: current year − year of first publication + 1)

Representative Publications

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1 February 2021 in Journal of Clinical Sleep Medicine
Verified
Journal Article
Closed access
JEJack D. Edinger
Jack D. Edinger
JAJ. Todd Arnedt
J. Todd Arnedt
Suzanne M BertischSuzanne M Bertisch
Suzanne M Bertisch
CCColleen E. Carney
Colleen E. Carney
JHJohn J. Harrington
John J. Harrington
JMJennifer L. Martin
Jennifer L. Martin
Introduction:This guideline establishes clinical practice recommendations for the use of behavioral and psychological treatments for chronic insomnia disorder in adults.Methods:The American Academy...

7 March 2018 in SLEEP
Verified
Journal Article
Closed access
PDF file
Suzanne M BertischSuzanne M Bertisch
Suzanne M Bertisch
BPBenjamin D Pollock
Benjamin D Pollock
MMMurray A Mittleman
Murray A Mittleman
DBDaniel J Buysse
Daniel J Buysse
LBLydia A Bazzano
Lydia A Bazzano
SRSusan Redline
Susan Redline
To quantify the association between insomnia/poor sleep with objective short sleep and incident cardiovascular disease (CVD) and mortality in the general population. We conducted a time-to-event analysis of Sleep Heart Health Study data. Questionnaires and at-home polysomnography were performed between 1994 -1998. Participants were followed for a median 11.4 years (Q1-Q3, 8.8-12.4 years) until death or last contact. The primary exposure was insomnia or poor sleep with short sleep defined as: difficulty falling asleep, difficulty returning to sleep, early morning awakenings, or sleeping pill use, 16-30 nights/month; and total sleep <6 hours on polysomnography (PSG). We used proportional hazards models to estimate the association between insomnia/poor sleep with short sleep and CVD, as well as all-cause mortality. Among 4,994 participants (mean age 64.0 ± 11.1 years), 14.1% reported insomnia or poor sleep, of which 50.3% slept <6 hours. Among 4,437 CVD-free participants at baseline, we observed 818 incident CVD events. After propensity-adjustment, there was a 29% higher risk of incident CVD in the insomnia/poor sleep with short sleep group compared with the reference group (HR, 1.29, 95% CI, 1.00, 1.66), but neither the insomnia/poor sleep only nor short sleep only groups were associated with higher incident CVD. Insomnia/poor sleep with objective short sleep was not significantly associated with all-cause mortality (HR, 1.07, 95% CI, 0.86, 1.33). Insomnia/poor sleep with PSG-short sleep was associated with higher risk of incident CVD. Future studies should evaluate the impact of interventions to improve insomnia with PSG-short sleep on CVD.

1 February 2021 in Journal of Clinical Sleep Medicine
Verified
Journal Article
Closed access
JEJack D. Edinger
Jack D. Edinger
JAJ. Todd Arnedt
J. Todd Arnedt
Suzanne M BertischSuzanne M Bertisch
Suzanne M Bertisch
CCColleen E. Carney
Colleen E. Carney
JHJohn J. Harrington
John J. Harrington
JMJennifer L. Martin
Jennifer L. Martin
Introduction:The purpose of this systematic review is to provide supporting evidence for a clinical practice guideline on the use of behavioral and psychological treatments for chronic insomnia dis...

1 February 2014 in SLEEP
Verified
Journal Article
Closed access
Suzanne M BertischSuzanne M Bertisch
Suzanne M Bertisch
SHShoshana J. Herzig
Shoshana J. Herzig
JWJohn W. Winkelman
John W. Winkelman
CBCatherine Buettner
Catherine Buettner
S and full research articles published in SLEEP, the official journal of the APSS. Sleep and sleep disorder scientific findings.

24 July 2008 in Journal of General Internal Medicine
Verified
Journal Article
Closed access
PDF file
GBGurjeet S. Birdee
Gurjeet S. Birdee
ALAnna T. Legedza
Anna T. Legedza
RSRobert B. Saper
Robert B. Saper
Suzanne M BertischSuzanne M Bertisch
Suzanne M Bertisch
DEDavid M. Eisenberg
David M. Eisenberg
RPRussell S. Phillips
Russell S. Phillips

17 May 2016 in Brain
Verified
Journal Article
Open access
PDF file
RNRodrigo Noseda
Rodrigo Noseda
CBCarolyn A. Bernstein
Carolyn A. Bernstein
RNRony-Reuven Nir
Rony-Reuven Nir
ALAlice J. Lee
Alice J. Lee
AFAnne B. Fulton
Anne B. Fulton
RBRami Burstein
Rami Burstein
Migraine headache is uniquely exacerbated by light. Using psychophysical assessments in patients with normal eyesight we found that green light exacerbates migraine headache significantly less than white, blue, amber or red lights. To delineate mechanisms, we used electroretinography and visual evoked potential recording in patients, and multi-unit recording of dura- and light-sensitive thalamic neurons in rats to show that green activates cone-driven retinal pathways to a lesser extent than white, blue and red; that thalamic neurons are most responsive to blue and least responsive to green; and that cortical responses to green are significantly smaller than those generated by blue, amber and red lights. These findings suggest that patients’ experience with colour and migraine photophobia could originate in cone-driven retinal pathways, fine-tuned in relay thalamic neurons outside the main visual pathway, and preserved by the cortex. Additionally, the findings provide substrate for the soothing effects of green light.