Ming Tang

Prof. Ming Tang

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Research Focus & Metrics

Academic Career Length

18Years (2009-Present)

(Calculated once Scilit publication data is synced: current year − year of first publication + 1)

Representative Publications

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8 February 2017 in Reports on Progress in Physics
Verified
Journal Article
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WWWei Wang
Wei Wang
MTMing Tang
Ming Tang
H. Eugene StanleyH. Eugene Stanley
H. Eugene Stanley
LBLidia A Braunstein
Lidia A Braunstein
Models of epidemic spreading on complex networks have attracted great attention among researchers in physics, mathematics, and epidemiology due to their success in predicting and controlling scenarios of epidemic spreading in real-world scenarios. To understand the interplay between epidemic spreading and the topology of a contact network, several outstanding theoretical approaches have been developed. An accurate theoretical approach describing the spreading dynamics must take both the network topology and dynamical correlations into consideration at the expense of increasing the complexity of the equations. In this short survey we unify the most widely used theoretical approaches for epidemic spreading on complex networks in terms of increasing complexity, including the mean-field, the heterogeneous mean-field, the quench mean-field, dynamical message-passing, link percolation, and pairwise approximation. We build connections among these approaches to provide new insights into developing an accurate theoretical approach to spreading dynamics on complex networks.

29 May 2014 in Scientific Reports
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Journal Article
Open access
WWWei Wang
Wei Wang
MTMing Tang
Ming Tang
HYHui Yang
Hui Yang
YDYounghae Do
Younghae Do
YLYing-Cheng Lai
Ying-Cheng Lai
GLGyuwon Lee
Gyuwon Lee
The spread of disease through a physical-contact network and the spread of information about the disease on a communication network are two intimately related dynamical processes. We investigate the asymmetrical interplay between the two types of spreading dynamics, each occurring on its own layer, by focusing on the two fundamental quantities underlying any spreading process: epidemic threshold and the final infection ratio. We find that an epidemic outbreak on the contact layer can induce an outbreak on the communication layer and information spreading can effectively raise the epidemic threshold. When structural correlation exists between the two layers, the information threshold remains unchanged but the epidemic threshold can be enhanced, making the contact layer more resilient to epidemic outbreak. We develop a physical theory to understand the intricate interplay between the two types of spreading dynamics.

6 May 2015 in Scientific Reports
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Journal Article
Open access
YLYing Liu
Ying Liu
MTMing Tang
Ming Tang
Tao ZhouTao Zhou
Tao Zhou
YDYounghae Do
Younghae Do
Identifying the most influential spreaders is an important issue in understanding and controlling spreading processes on complex networks. Recent studies showed that nodes located in the core of a network as identified by thek-shell decomposition are the most influential spreaders. However, through a great deal of numerical simulations, we observe that not in all real networks do nodes in high shells are very influential: in some networks the core nodes are the most influential which we call true core, while in others nodes in high shells, even the innermost core, are not good spreaders which we call core-like group. By analyzing thek-core structure of the networks, we find that the true core of a network links diversely to the shells of the network, while the core-like group links very locally within the group. For nodes in the core-like group, thek-shell index cannot reflect their location importance in the network. We further introduce a measure based on the link diversity of shells to effectively distinguish the true core and core-like group and identify core-like groups throughout the networks. Our findings help to better understand the structural features of real networks and influential nodes.

27 July 2015 in Physical Review E
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WWWei Wang
Wei Wang
MTMing Tang
Ming Tang
HZHai-Feng Zhang
Hai-Feng Zhang
YLYing-Cheng Lai
Ying-Cheng Lai
A key ingredient in social contagion dynamics is reinforcement, as adopting a certain social behavior requires verification of its credibility and legitimacy. Memory of nonredundant information plays an important role in reinforcement, which so far has eluded theoretical analysis. We first propose a general social contagion model with reinforcement derived from nonredundant information memory. Then, we develop a unified edge-based compartmental theory to analyze this model, and a remarkable agreement with numerics is obtained on some specific models. We use a spreading threshold model as a specific example to understand the memory effect, in which each individual adopts a social behavior only when the cumulative pieces of information that the individual received from his or her neighbors exceeds an adoption threshold. Through analysis and numerical simulations, we find that the memory characteristic markedly affects the dynamics as quantified by the final adoption size. Strikingly, we uncover a transition phenomenon in which the dependence of the final adoption size on some key parameters, such as the transmission probability, can change from being discontinuous to being continuous. The transition can be triggered by proper parameters and structural perturbations to the system, such as decreasing individuals' adoption threshold, increasing initial seed size, or enhancing the network heterogeneity.

6 October 2014 in Physical Review E
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Journal Article
Closed access
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WWWei Wang
Wei Wang
MTMing Tang
Ming Tang
HZHai-Feng Zhang
Hai-Feng Zhang
HGHui Gao
Hui Gao
YDYounghae Do
Younghae Do
ZLZong-Hua Liu
Zong-Hua Liu
The spread of disease on complex networks has attracted wide attention in the physics community. Recent works have demonstrated that heterogeneous degree and weight distributions have a significant influence on the epidemic dynamics. In this study, a novel edge-weight-based compartmental approach is developed to estimate the epidemic threshold and epidemic size (final infected density) on networks with general degree and weight distributions, and a remarkable agreement with numerics is obtained. Even in complex networks with the strong heterogeneous degree and weight distributions, this approach is used. We then propose an edge-weight-based removal strategy with different biases and find that such a strategy can effectively control the spread of epidemic when the highly weighted edges are preferentially removed, especially when the weight distribution of a network is extremely heterogenous. The theoretical results from the suggested method can accurately predict the above removal effectiveness.

17 August 2015 in Scientific Reports
Verified
Journal Article
Open access
YLYing Liu
Ying Liu
MTMing Tang
Ming Tang
Tao ZhouTao Zhou
Tao Zhou
YDYounghae Do
Younghae Do
Recent study shows that the accuracy of thek-shell method in determining node coreness in a spreading process is largely impacted due to the existence of core-like group, which has a largek-shell index but a low spreading efficiency. Based on the analysis of the structure of core-like groups in real-world networks, we discover that nodes in the core-like group are mutually densely connected with very few out-leaving links from the group. By defining a measure of diffusion importance for each edge based on the number of out-leaving links of its both ends, we are able to identify redundant links in the spreading process, which have a relatively low diffusion importance but lead to form the locally densely connected core-like group. After filtering out the redundant links and applying thek-shell method to the residual network, we obtain a renewed corenessksfor each node which is a more accurate index to indicate its location importance and spreading influence in the original network. Moreover, we find that the performance of the ranking algorithms based on the renewed coreness are also greatly enhanced. Our findings help to more accurately decompose the network core structure and identify influential nodes in spreading processes.