Background: Although data demonstrate the ability of the menisci to transmit load, they do not contribute to the primary stability of the knee. In absence of ACL, the menisci have shown to enhance the knee's stability in AP, varus-valgus, internal-external direction in vitro. The importance of addressing meniscal pathology associated with anterior cruciate ligament (ACL) insufficiency stems from the increased incidence of meniscal tear with chronic instability. Most of the studies showed Lateral meniscus injury with acute ACL tear.
Checkrein deformity may occur after leg or ankle fractures. It is a dynamic flexion deformity of the big toe after planter flextion of the ankle that disappear when the ankle is dorsiflexed. It is believed to be the result of Tethering or entrapment of the flexor hallucis longus (FHL) tendon in scar tissue or callus at the fracture site,1,2 It was also suggested that it results from contracture of the FHL muscles due to a subclinical or missed compartment syndrome.3 Checkrein deformity is usually noticed during follow up in clinic and it may not respond to conservative management like stretching exercises. Surgical treatment is best done at midfoot by FHL tendon lengthening over lengthening at the fracture site.
Isolated paediatric femoral fracture is common and can be missed due to many Factors such as patient related, clinical, technical and radiological error. Careful listening to patient complain and examination along with careful look up and emphasising in radiological evaluation used for diagnosis. Prophylactic hip spica is an option in such condition for short period and callus might show up.
Background/Objectives: The distorted vertebral body has been studied in scoliosis; however, there is little knowledge about the difference between neuromuscular and idiopathic scoliosis. This study aimed to investigate the vertebral body morphology in patients with spastic quadriplegic cerebral palsy and scoliosis (CP scoliosis) and compare them with those of apex- and Cobb angle-matched patients with adolescent idiopathic scoliosis (AIS). Methods: Thirty-four patients with CP scoliosis and thirty-two patients with AIS were included. The pedicle diameter, chord length, and vertebral body rotation were evaluated at one level above the apex, one level below the apex, and at the apex using a reconstructed computed tomography scan. The apex of the curve and Cobb angle were too diverse between patients with CP scoliosis or AIS. Eighteen patients were matched in each group according to the apex and Cobb angle (within 5-degree differences) of the major curve, and compared between matched groups (mCPscoliosis vs. mAIS). Results: In the comparison of the apex and Cobb angle-matched groups, there was no statistical difference in the Cobb angle between mCPscoliosis (80.7 ± 13.8 degrees) and mAIS (78.6 ± 13.6 degrees, p = 0.426), and the vertebral body rotation (25.4 ± 15.4° in mCPscoliosis vs. 24.4 ± 6.5° in mAIS, p = 0.594). There was no difference in the pedicle diameters of either the convex (3.6 ± 1.1 mm in mCPscoliosis vs. 3.3 ± 1.2 mm in mAIS, p = 0.24) or concave side (3.1 ± 1.2 mm in mCPscoliosis vs. 2.7 ± 1.6 mm in mAIS, p = 0.127). However, the patients in the mCPscoliosis group were younger (12.7 ± 2.5 years vs. 14.6 ± 2.4 years, p = 0.001), and the chord length was shorter on the convex (38.0 ± 5.0 mm vs. 40.4 ± 4.9 mm, p = 0.025) and concave (37.7 ± 5.2 mm vs. 40.3 ± 4.7 mm, p = 0.014) sides compared with those of the mAIS group. Conclusions: With a similar apex and Cobb angle, the vertebral body rotation and pedicle diameter in patients with CP scoliosis were comparable to those with AIS; however, the chord length was shorter in CP scoliosis. For the selection of the pedicle screw in CP scoliosis, the length of the pedicle screw should be more considered than the diameter.