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Dr. Chunling Huang

Kolling Institute, University of Sydney

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Dr Huang is a research fellow in the renal lab at Kolling Institute, Sydney Medical School, University of Sydney. She completed a medical degree and a master’s degree in China and was a visiting research associate in Vrije Universiteit Medical Center, Amsterdam from 2006-2008. In 2014, Dr Huang was awarded the PhD degree in University of Sydney with 22 publications, 10 of which were as first author. Her PhD study was to examine the role of blocking the KCa3.1 channel to prevent or retard the progression of diabetic kidney disease. This led to a successful Juvenile Diabetes Research Foundation International (JDRF) grant under the Novel Therapeutics Program as well as a provisional patent. Dr Huang has expertise in diabetic kidney disease, animal models of diabetic nephropathy and kidney fibrosis. Her research focuses on the novel therapeutic targets for diabetic kidney disease and the underling mechanisms including inflammation, fibrosis, fibroblast activation and autophagy/mitophagy.

Research Keywords & Expertise

Chronic Kidney Disease
Kidney Disease
Medical Research
diabetic kidney diseas...
kidney fibrosis

Short Biography

Dr Huang is a research fellow in the renal lab at Kolling Institute, Sydney Medical School, University of Sydney. She completed a medical degree and a master’s degree in China and was a visiting research associate in Vrije Universiteit Medical Center, Amsterdam from 2006-2008. In 2014, Dr Huang was awarded the PhD degree in University of Sydney with 22 publications, 10 of which were as first author. Her PhD study was to examine the role of blocking the KCa3.1 channel to prevent or retard the progression of diabetic kidney disease. This led to a successful Juvenile Diabetes Research Foundation International (JDRF) grant under the Novel Therapeutics Program as well as a provisional patent. Dr Huang has expertise in diabetic kidney disease, animal models of diabetic nephropathy and kidney fibrosis. Her research focuses on the novel therapeutic targets for diabetic kidney disease and the underling mechanisms including inflammation, fibrosis, fibroblast activation and autophagy/mitophagy.