Alexa M. L'Insalata

Ms. Alexa M. L'Insalata

Psychology, University of Kansas, Lawrence, Indiana, USA
orcid

Research Focus & Metrics

Academic Career Length

4Years (2023-Present)

(Calculated once Scilit publication data is synced: current year − year of first publication + 1)

Representative Publications

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4 October 2023 in Obesity
Verified
Journal Article
Closed access
CSCassandra A. Sutton
Cassandra A. Sutton
MSMatthew Stratton
Matthew Stratton
ALAlexa M. L'Insalata
Alexa M. L'Insalata
TFTera Fazzino
Tera Fazzino

18 January 2024 in International Journal of Eating Disorders
Verified
Journal Article
Closed access
SJSarah Johnson‐Munguia
Sarah Johnson‐Munguia
ABAngeline R. Bottera
Angeline R. Bottera
IVIrina Vanzhula
Irina Vanzhula
KFKelsie T. Forbush
Kelsie T. Forbush
SGSara R. Gould
Sara R. Gould
SRSamiya Rasheed
Samiya Rasheed

16 April 2025 in Nutrients
Verified
Journal Article
Open access
PDF file
JBJoseph S. Bellitti
Joseph S. Bellitti
ALAlexa M. L'Insalata
Alexa M. L'Insalata
TFTera Fazzino
Tera Fazzino
Background: High delay discounting (DD), or the tendency to prefer immediate rewards over larger delayed rewards, has been associated with health risk behaviors. This study examined the DD of hyper-palatable foods (HPFs) and money, and their associations with eating motives and binge eating behavior. Methods: An adult sample (N = 296) completed a DD task with single- and cross-commodity conditions with rewards of money and/or HPF (e.g., HPF now vs. HPF later; HPF now vs. money later). Regression models tested the association between DD, and eating motives and binge eating. Results: DD in the HPF now vs. money later condition was positively associated with the reward enhancement motive (β = 0.291; p = 0.008) and binge eating behavior (β = 0.041; p = 0.017). There were no other significant associations. Conclusions: Decisional impulsivity specific to HPFs (and not decisional impulsivity in general) may be associated with reward-motivated eating and binge eating behavior.

12 August 2024 in International Journal of Eating Disorders
Verified
Journal Article
Closed access
MTMarianna L. Thomeczek
Marianna L. Thomeczek
KFKelsie T. Forbush
Kelsie T. Forbush
YCYiyang Chen
Yiyang Chen
SNSonakshi Negi
Sonakshi Negi
SJSarah Johnson‐Munguia
Sarah Johnson‐Munguia
JMJacquelyn McDonald
Jacquelyn McDonald

21 October 2026 in European Eating Disorders Review
Verified
Journal Article
Open access
SNSonakshi Negi
Sonakshi Negi
ABAngeline R. Bottera
Angeline R. Bottera
MTMarianna L. Thomeczek
Marianna L. Thomeczek
ELElizabeth A. Li
Elizabeth A. Li
ALAlexa M. L'Insalata
Alexa M. L'Insalata
KFKelsie T. Forbush
Kelsie T. Forbush
Compensatory eating disorder (CED) is a newly proposed ‘other specified feeding and eating disorder’ that is characterised by recurrent non‐purging compensatory behaviours, overvaluation of weight and shape, and the absence of objective binge eating, low weight, or recent significant weight loss. Although CED has been long documented in research, it remains unclear whether CED represents a distinct clinical diagnosis or a variant presentation of extant eating disorders. In this commentary, we situate CED within the broader eating disorders landscape and call for greater diagnostic clarity. We also provide five concrete future directions that can inform the appropriate classification of CED, including: (1) clarifying core diagnostic features of CED, (2) leveraging statistical models to inform CED classification, (3) establishing CED's longitudinal and prognostic validity, (4) incorporating clinician and patient perspectives on the adoption of CED diagnostic criteria, and (5) weighing risks and benefits of adding a new clinical diagnosis. These guidelines may help our field answer a novel research question that has implications for clinical practice: Is Compensatory Eating Disorder a missing diagnosis in the eating‐disorder nomenclature or is it a misclassified one?

ALAlexa M. L'Insalata
Alexa M. L'Insalata
JGJeffrey Girard
Jeffrey Girard
TFTera Fazzino
Tera Fazzino
Research supports the premise that greater substance use is associated with fewer sources of environmental reinforcement. However, it remains unclear whether types of environmental reinforcement (e.g., social or work) may differentially influence use. This study tested the association between types of environmental reinforcement and engagement in multiple health risk behaviors (alcohol use, binge eating, and nicotine use). Cross-sectional data were collected from a general population sample of US adults (N = 596). The Pleasant Events Schedule (PES) was used to measure sources of reinforcement. Exploratory structural equation modeling (ESEM) characterized different areas of environmental reinforcement and correlations with alcohol consumption, binge eating, and nicotine use. A four-factor structure of the PES demonstrated a conceptually cohesive model with acceptable fit and partial strict invariance. Social-related reinforcement was positively associated with alcohol consumption (β = 0.30, p < 0.001) and binge eating (β = 0.26, p < 0.001). Work/school-related reinforcement was negatively associated with binge eating (β = −0.14, p = 0.006). No areas of reinforcement were significantly associated with nicotine use (p values = 0.069 to 0.755). Social-related activities may be associated with engagement in multiple health risk behaviors (more binge eating and alcohol use), whereas work/school-related activities may be preventative against binge eating. Understanding these relationships can inform prevention efforts targeting health risk behaviors.