Thyroid disorders are common in patients with autoimmune diseases such as rheumatoid arthritis (RA). Both conditions present significant public health concerns due to their impact on quality of life and increased mortality. The aim of this study was to assess the prevalence of thyroid abnormalities in and investigate their influence on rheumatoid arthritis characteristics. This was a case–control study involving adult female patients with RA and age-matched healthy controls. RA disease activity was evaluated using the Disease Activity Score (DAS28), and functional impact was assessed using the Health Assessment Questionnaire (HAQ). Serum thyroid function tests were performed, including thyroid-stimulating hormone (TSH), free thyroxine (FT4), and antithyroid antibodies (TAAs), including anti-thyroglobulin antibodies (TgAb), anti-thyroperoxidase antibodies (TPOAb), and TSH receptor antibodies (TRAb). The study included 58 female RA patients, with a mean age of 52 ± 14.2 years. The median disease duration was 10.41 years. The median DAS28-CRP was 4.03, and the median HAQ was 1.34. The median FT4 and TSH levels were 15.01 [13.48;16.71] and 1.42 [0.91;2.26], respectively. Thyroid dysfunction was identified in 19% of the participants, with hypothyroidism being the most common disorder (17%). Hyperthyroidism was observed in 2% of patients. Antithyroid antibodies were positive in 15.5% (n = 9) of participants, with TPOAb present in 6 patients (10.3%), TgAb in 3 patients (5.2%), and TRAb in 2 patients (3.4%). No statistically significant association was found between thyroid status and RA disease activity, functional impact, or serological status. Despite the lack of correlation, the high prevalence of thyroid dysfunction underscores the importance of regular thyroid screening to optimize management and prognosis in RA patients.

Case presentation An 81-year-old female patient with no medical history presented with a 1-day history of inflammatory upper cervical spine and occipital region pain. Physical examination showed restricted rotation in the neck and normal temporal pulses. Neurological examination was unremarkable. Her body temperature was 38.2 °C. Laboratory tests showed a high C-reactive protein level (364 mg/L, normal value N < 8) and erythrocyte sedimentation rate (86 mm, N < 25) alongside a mildly elevated white cell count (10.5 × 10 9 /L, N < 10). Urine analysis and blood cultures were negative. A cervical computed tomography (CT) scan was performed revealing a crown-like calcification surrounding the odontoid process and no sign of an infectious process (Fig. 1).

Introduction: Parsonage-Turner Syndrome is an uncommon cause of shoulder pain Case Representation: Herein, we present the case of a male in his 40s, who was presented with a 3-month history of acute onset of intense shoulder pain, which decreased rapidly leaving behind a residual upper limb weakness. The diagnosis of Parsonage-Turner Syndrome following COVID-19 vaccination was made based on electroneuromyography and magnetic resonance imaging findings. The patient responded well to analgesics and rehabilitation. Conclusion: A better knowledge of this disease and early recognition are crucial to prevent unnecessary tests and interventions.

Background: Amyloid arthropathy is characterized by the deposition of misfolded proteins in the joints and soft tissues. It is often a manifestation of light chain amyloidosis. The ultrasonographic features of amyloid arthropathy are solely reported in the literature. Case Presentation: Herein, we present the case of a 70-year-old patient who was diagnosed with light chain amyloidosis. He reported chronic joint pain, bilateral carpal tunnel syndrome, and an inguinal mass. Ultrasound examination revealed tenosynovitis of the flexor digitorum tendons, the extensor carpi ulnaris tendon, and the long head of the biceps tendon, along with synovitis in the wrists, elbows, and shoulders, as well as knee joint effusion. The synovial thickening with heterogeneous echogenic material suggested amyloid deposition. Conclusion: This case underscored key ultrasonographic features of amyloid arthropathy, including synovial thickening with heterogeneous echogenic deposits, tenosynovitis, and subacromialsubdeltoid bursa involvement. Unlike rheumatoid arthritis, amyloidosis lacked erosions and power Doppler signal, highlighting imaging distinctions. The hypoechoic inguinal amyloidoma with calcifications further aligned with amyloid deposition. Although amyloidosis shares certain clinical features with dialysis-related β2-microglobulin amyloidosis (e.g., carpal tunnel syndrome, shoulder deposits), AL amyloidosis may exhibit unique patterns, such as diffuse synovial infiltration without hyperemia. However, ultrasound’s non-specificity necessitates histopathological confirmation. While systemic amyloidosis requires pathological confirmation, ultrasonography provides a rapid, cost-effective tool for early diagnostic guidance.