Matthew R. Avenarius, Ph.D.,
is an Assistant Professor, Clinical, in the Department of Pathology and serves as
Associate Director of the Molecular Pathology Laboratory (Genetics). Dr.
Avenarius earned his PhD in Human Genetics from the University of Michigan and
did his postdoctoral research in the Barr-Gillespie Lab at Oregon Health &
Science University. He continued his training at Nationwide Children’s Hospital,
where he completed his clinical molecular genetics and genomics and clinical
cytogenetics and genomics fellowship. Dr. Avenarius is a Diplomate of the
American Board of Medical Genetics and Genomics with subspecialty certification
in the areas of clinical molecular genetics and cytogenetics. As Associate
Director of the Molecular Pathology Laboratory, Dr. Avenarius is responsible
for overseeing the development, validation, and implementation of new
diagnostic assays and is active in clinical case sign-out. Dr. Avenarius’s
research interests include the genetics of hereditary hearing loss and spinal
muscular atrophy.
Matthew R. Avenarius, Ph.D.,
is an Assistant Professor, Clinical, in the Department of Pathology and serves as
Associate Director of the Molecular Pathology Laboratory (Genetics). Dr.
Avenarius earned his PhD in Human Genetics from the University of Michigan and
did his postdoctoral research in the Barr-Gillespie Lab at Oregon Health &
Science University. He continued his training at Nationwide Children’s Hospital,
where he completed his clinical molecular genetics and genomics and clinical
cytogenetics and genomics fellowship. Dr. Avenarius is a Diplomate of the
American Board of Medical Genetics and Genomics with subspecialty certification
in the areas of clinical molecular genetics and cytogenetics. As Associate
Director of the Molecular Pathology Laboratory, Dr. Avenarius is responsible
for overseeing the development, validation, and implementation of new
diagnostic assays and is active in clinical case sign-out. Dr. Avenarius’s
research interests include the genetics of hereditary hearing loss and spinal
muscular atrophy.