Stela Mirla da Silva da Silva Felipe

Dr. Stela Mirla da Silva da Silva Felipe


Research Focus & Metrics

Academic Career Length

4Years (2023-Present)

(Calculated once Scilit publication data is synced: current year − year of first publication + 1)

Representative Publications

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20 November 2023 in Pathogens
Verified
Journal Article
Open access
PDF file
EPEric Pereira
Eric Pereira
SFStela Mirla da Silva da Silva Felipe
Stela Mirla da Silva da Silva Felipe
RFRaquel Martins de Freitas
Raquel Martins de Freitas
José Ednésio da Cruz FreireJosé Ednésio da Cruz Freire
José Ednésio da Cruz Freire
AOAntonio Edson Rocha Oliveira
Antonio Edson Rocha Oliveira
Vania CeccattoVania Ceccatto
Vania Ceccatto
The COVID-19 disease, caused by the Severe Acute Respiratory Syndrome Coronavirus 2 (SARS-CoV-2), emerged in late 2019 and rapidly spread worldwide, becoming a pandemic that infected millions of people and caused significant deaths. COVID-19 continues to be a major threat, and there is a need to deepen our understanding of the virus and its mechanisms of infection. To study the cellular responses to SARS-CoV-2 infection, we performed an RNA sequencing of infected vs. uninfected Calu-3 cells. Total RNA was extracted from infected (0.5 MOI) and control Calu-3 cells and converted to cDNA. Sequencing was performed, and the obtained reads were quality-analyzed and pre-processed. Differential expression was assessed with the EdgeR package, and functional enrichment was performed in EnrichR for Gene Ontology, KEGG pathways, and WikiPathways. A total of 1040 differentially expressed genes were found in infected vs. uninfected Calu-3 cells, of which 695 were up-regulated and 345 were down-regulated. Functional enrichment analyses revealed the predominant up-regulation of genes related to innate immune response, response to virus, inflammation, cell proliferation, and apoptosis. These transcriptional changes following SARS-CoV-2 infection may reflect a cellular response to the infection and help to elucidate COVID-19 pathogenesis, in addition to revealing potential biomarkers and drug targets.