Luxação intrusiva é o deslocamento do dente em seu alvéolo, na direção axial, favorecido pela baixa resiliência do osso alveolar. Clinicamente, esse trauma pode apresentar: Grau I (intrusão parcial leve, em que mais de 50% da coroa é visível), Grau II (intrusão parcial moderada, em que menos de 50% da coroa é visível), Grau III (intrusão severa ou completa da coroa). O objetivo desse estudo foi relatar um caso de luxação intrusiva Grau III em uma criança, discutido aspectos acerca de seu diagnóstico, modalidade terapêutica e o acompanhamento do caso. Um paciente de 15 meses de idade, compareceu à Bebê Clínica da Faculdade de Odontologia de Araçatuba uma semana após sofrer um trauma. O exame clínico revelou gengivas inchadas além da ausência do elemento 51. Após exame radiográfico, a intrusão completa do dente 51 foi confirmada, com o deslocamento do ápice da raiz para a direção vestibular. O tratamento foi aguardar a reerupção espontânea do dente; além de orientações quanto ao alívio oclusal (controle do uso de bicos, e hábitos bucais como onicofagia) e sobre higiene bucal. Após três meses de acompanhamento, a erupção total do dente 51 foi observada, e a gengiva assim como o ápice do dente traumatizado apresentavam aspecto normais. Pode-se concluir que controle clínico e radiográfico, especialmente em casos de injúrias traumáticas em dentes decíduos, além de monitorar o caso até a erupção do dente permanente, detectando possíveis sequelas no dente sucessor e podem se apresentar como alternativas eficientes para o tratamento da luxação intrusiva em dentes com rizogênese incompleta.

Although the anticaries properties of phosphate-based compounds have been extensively investigated in recent years, their potential cytotoxic effects remain underexplored. This study evaluated the cytotoxicity of solutions containing sodium trimetaphosphate (TMP), sodium hexametaphosphate (HMP), or calcium glycerophosphate (CaGP). NIH/3T3 fibroblasts were cultured in Dulbecco’s Modified Eagle Medium (DMEM) supplemented with 10% fetal bovine serum and maintained at 37 °C, 100% humidity, and 5% CO₂. The cells were seeded in 96-well plates at a density of 10⁴ cells per well and incubated for 24 h. Subsequently, different dilutions of 10% TMP, HMP, or CaGP solutions were applied to the cells. Cell viability was assessed at 24/48 h using the MTT assay. The data were subjected to two-way ANOVA and Fisher’s LSD test. Spearman’s rank correlation was performed. HMP dilutions led to significantly lower cell viability compared to the other compounds, regardless of the incubation period. TMP maintained higher cell viability from 1/8 dilution onwards, regardless of the incubation time. For CaGP, an increase in cell viability was observed at 1/8 dilution after 24 h. In conclusion, TMP and CaGP demonstrated reduced cytotoxicity at higher dilutions compared to HMP, suggesting their potential as promising candidates for the development of novel biomaterials.

Calcium-modified cyclophosphates may enhance protection against enamel demineralization. This study synthesized and characterized calcium cyclohexaphosphate (CaCHP) and evaluated its concentration-dependent protective effect against initial enamel demineralization under erosive conditions in vitro. CaCHP was obtained from sodium cyclohexaphosphate (NaCHP) by ion-exchange resin and characterized by SEM, EDS, XRD, and FTIR. Sound and softened bovine enamel blocks (n = 12/group) were treated with toothpaste slurries prepared in human saliva containing 0.25%, 0.5%, or 1% NaCHP; 0.25%, 0.5%, or 1% CaCHP; 0.24% NaF; or placebo. Blocks underwent four consecutive citric acid challenges (pH 3.5, 1 min), interspersed with surface hardness measurements. Data were analyzed by three-way ANOVA and Tukey tests ( p < 0.05). Enamel surfaces were also examined by SEM/EDS. Characterization confirmed Na replacement by Ca within a cyclic structure and greater crystallinity for CaCHP than NaCHP. All groups showed decreased hardness during acid challenges. However, softened enamel treated with NaF or cyclophosphates showed a smaller decrease in surface hardness than sound enamel ( p < 0.05). Placebo showed the greatest decrease in hardness ( p < 0.05), regardless of initial enamel condition. In sound enamel, 1% CaCHP and 0.24% NaF showed comparable protection through the third acid challenge, but neither differed from placebo after the fourth challenge. In softened enamel, 1% CaCHP remained comparable to 0.24% NaF and significantly more protective than placebo and NaCHP after the fourth challenge ( p < 0.001). In conclusion, CaCHP was synthesized from NaCHP, and 1% CaCHP provided protection comparable to 0.24% NaF, with persistence depending on enamel condition.

The aim of this study was to evaluate fluoride release, radiopacity and rugosity in glass ionomer surface of four restorative glass ionomer cements subjected to pH cycling. Six specimens from each experimental group (Ketac N100, Vitremer, Riva Light Cure, Fuji IX, Riva Self Cure) were first immersed in 2mL of demineralizing solutions (De) for six hours and then placed in the remineralizing solution (Re) for eighteen hours, this cycle repeated for 7 days. Radiopacity and roughness were measured at the beginning and end of cycling. The fluoride released in the Des- and Re- solutions was analyzed at 24-hour intervals over the 7 days. The data were submitted to two-way ANOVA and Fisher's PLSD test (p<0.05). The Ketac N100 group showed lower roughness and greater radiopacity before and after pH cycling, when compared to the other groups (p<0.05). All glass ionomer cements evaluated showed greater fluoride release on the first day of cycling. The Riva Light Cure, Vitremer and Fuji IX groups had a higher mean fluoride release during the 7 days of cycling when compared to Ketac N100. It was concluded that Ketac N100 presented, before and after cycling, lower roughness and greater radiopacity when compared to the other groups. In addition, it presented fluoride released higher than Riva Light Cure (SDI-L) and Riva Self Cure (SDI-S), but similar to Vitremer (VIT) and Fuji IX (F9).