Background: Heart failure (HF) is an increasing burden in Sub-Saharan Africa, with coronary artery disease (CAD) emerging as a major cause. However, angiographic data in HF patients remain scarce in this region. Methods: This retrospective study included 169 HF patients who underwent coronary angiography between 2023 and 2025 at Mogadishu Somali Türkiye Training and Research Hospital. CAD severity was classified as minor, moderate, or severe (≥ 70% stenosis in epicardial arteries or ≥ 50% in the left main). Demographic, clinical, and echocardiographic data were analyzed. Logistic regression identified predictors of severe CAD. Results: The mean age was 54.3 ± 10.9 years, and 33.1% were male. CAD was present in 53.3% of patients, with severe CAD in 23.1%. Single-vessel disease was most frequent, followed by double-vessel, triple-vessel, and left main involvement. Based on SYNTAX scoring, 27.2% had low, 10.1% intermediate, and 6.5% high scores, indicating a subset with complex anatomy. On univariate analysis, diabetes (P < 0.001), dyslipidemia (P < 0.001), smoking (P = 0.03), and khat chewing (P = 0.03) were significantly associated with severe CAD. Patients with severe CAD also had lower ejection fraction (26.3% vs 29.8%, P = 0.01) and higher SYNTAX scores (34.7 vs 19.6, P < 0.001). In multivariate analysis, diabetes (OR = 7.44, 95% CI: 3.04– 18.17, P < 0.001) and smoking (OR = 2.61, 95% CI: 1.10– 6.23, P = 0.03) remained independent predictors. Conclusion: Severe CAD is common among HF patients in Somalia and is strongly associated with diabetes and smoking. Systematic ischemic evaluation and aggressive risk factor control should be prioritized to improve outcomes. This study provides one of the first angiographic datasets from the region, offering evidence to guide screening, optimize scarce diagnostic resources, and inform future multicenter research.

Objective: To evaluate clinical outcomes and identify predictors of mortality and endoleak after endovascular aneurysm repair (EVAR) and thoracic endovascular aortic repair (TEVAR) procedures. Methods: A retrospective cohort study of 164 EVAR and TEVAR patients (January 2000–December 2022) was conducted. Data on demographics, comorbidities, aneurysm size, and clinical outcomes were extracted. Statistical analysis included Pearson’s chi-square test, Student’s t -test, Mann–Whitney U test, and multivariate logistic regression. Model performance was assessed by receiver operating characteristic and Hosmer–Lemeshow tests. Results: Overall mortality was 7%, and endoleak incidence was 20.75%. Diabetes mellitus was an independent predictor of mortality [adjusted odds ratio (AOR) = 3.93; 95% CI: 1.51–10.24, P = 0.005], and aneurysm size (>7 cm) independently predicted endoleaks (AOR = 3.21; 95% CI: 1.28–8.06, P = 0.013). Physician specialty and procedure type did not significantly predict outcomes. Model discrimination (area under the curve = 0.96) and calibration (Hosmer–Lemeshow P = 0.400) were strong. Conclusion: Diabetes mellitus and aneurysm size (>7 cm) significantly influence clinical outcomes following EVAR and TEVAR, guiding risk stratification and patient management.

Introduction: Pulmonary embolism (PE) is a critical condition that can present with diverse and often non-specific electrocardiographic findings. The Aslanger pattern, identified in 2020, is an uncommon electrocardiography (ECG) finding typically associated with acute non-ST-segment elevation myocardial infarction (NSTEMI). Although this pattern is usually linked to myocardial infarction with significant coronary stenosis, we report a rare case where the Aslanger pattern was observed in a patient with acute pulmonary embolism. Case Presentation: A 52-year-old male with Parkinson’s disease presented with sudden chest pain, shortness of breath, sweating, and fatigue. Initial ECG showed Aslanger pattern, suggesting acute coronary syndrome. Despite elevated troponin I (1624 ng/mL) and normal coronary angiography, further investigation revealed right ventricular dilation and massive pulmonary embolism confirmed by CT angiography. The patient was treated with thrombolytics and anticoagulation, stabilizing over five days and discharged on rivaroxaban. Conclusion: The Aslanger pattern usually linked to myocardial infarction can also occur in acute pulmonary embolism. This case underscores the need for careful differential diagnosis and timely treatment to prevent complications associated with delayed care.

Yurdumuzun birçok bölgesinden ve değişik hastanelerden değerli uzmanlarımızın hazırladığı bu vakalarla kardiyoloji kitabı umarım sizlerin beğenisini kazanır, yazarların emeği yerini bulur ve daha sonraki baskıları veya yeni kitaplar için cesaret verici olur. Bu arada kitap ile ile ilgili eleştirilerinizi yazıp gönderirseniz ilerdeki baskılar için yol gösterici olur. Kitapta göreceğiniz gibi ağırlık girişimsel kardiyoloji vakalarına ait. Bu da kardiyologların en çok hangi alanlarda çalıştıklarını ve ilgilendiklerini gösteriyor. Umarım sonraki baskılarda girişimsel olmayan, özellikle görüntüleme teknikleri ile ilgili yazılar da olacaktır.

Background and Objectives: Left ventricular global longitudinal strain (LV GLS) has prognostic relevance in hypertrophic cardiomyopathy (HCM), but its incremental value for identifying nonsustained ventricular tachycardia (NSVT) documented on ambulatory monitoring is uncertain. We examined whether signed LV GLS was independently associated with documented NSVT and whether it improved a fixed clinical and conventional echocardiographic model. Materials and Methods: This retrospective, single-centre cross-sectional study included 201 patients with HCM. Transthoracic echocardiography was performed on the day that 24-h ambulatory electrocardiographic monitoring was initiated. A reference logistic regression model included age, sex, maximal septal thickness, maximal left ventricular outflow tract (LVOT) gradient, and ejection fraction; signed LV GLS was then added. Discrimination and calibration were internally validated using 2000 bootstrap samples with coefficient refitting. Six secondary mechanical measures were analysed with false discovery rate control. Results: NSVT was documented in 115 patients (57.2%). Median LV GLS was −11.3% [interquartile range, −14.3 to −9.0] with NSVT and −10.9% [−14.3 to −9.2] without NSVT (p = 0.819); the 0.4-percentage-point difference lies within the measurement variability of LV GLS, and the two distributions overlapped almost completely. The adjusted odds ratio (OR) for LV GLS per 1-percentage-point increase was 0.962 (95% confidence interval [CI], 0.884–1.047; p = 0.372). The apparent area under the curve was 0.645 (95% CI, 0.564–0.724) for the reference model and 0.644 (95% CI, 0.564–0.723) after adding LV GLS (paired difference, −0.001; 95% CI, −0.029 to 0.027; likelihood-ratio p = 0.370); optimism-corrected values were 0.606 and 0.598. In exploratory analyses, higher global radial strain was associated with lower odds of NSVT (OR per standard deviation, 0.619; 95% CI, 0.449–0.852; q = 0.020). Conclusions: LV GLS was not independently associated with documented NSVT and did not improve its discrimination beyond conventional clinical and echocardiographic variables. The exploratory radial strain association requires independent confirmation.