Exercise is impaired in hot, compared with moderate, conditions. The development of hyperthermia is strongly linked to the impairment and as a result various strategies have been investigated to combat this condition. This meta-analysis focused on the most popular strategy: cooling. Precooling has received the most attention but recently cooling applied during the bout of exercise has been investigated and both were reviewed. We conducted a literature search and retrieved 28 articles which investigated the effect of cooling administered either prior to (n=23) or during (n=5) an exercise test in hot (wet bulb globe temperature >26°C) conditions. Mean and weighted effect size (Cohen's d) were calculated. Overall, precooling has a moderate (d=0.73) effect on subsequent performance but the magnitude of the effect is dependent on the nature of the test. Sprint performance is impaired (d=−0.26) but intermittent performance and prolonged exercise are both improved following cooling (d=0.47 and d=1.91, respectively). Cooling during exercise has a positive effect on performance and capacity (d=0.76). Improvements were observed in studies with and without cooling-induced physiological alterations, and the literature supports the suggestion of a dose–response relationship among cooling, thermal strain and improvements in performance and capacity. In summary, precooling can improve subsequent intermittent and prolonged exercise performance and capacity in a hot environment but sprint performance is impaired. Cooling during exercise also has a positive effect on exercise performance and capacity in a hot environment.

Etitive soccer match. Seven semiprofessional soccer players participated in a competitive league match. Blood and saliva samples were obtained 1 hour before kick off and immediately postmatch. Global positioning system equipment was used to measure heart rate and activity profile data throughout the match. Percentage increase in creatine kinase (CK) and myoglobin (MYO) concentrations was correlated with the number of sprints performed during the match (r = 0.88, p = 0.019; r = 0.75, p = 0.047, respectively). Creatine kinase increased by 84% (p = 0.17) from prematch to postmatch, whereas MYO increased by 238% (p = 0.05). Players performed 39 ± 18 sprints during the course of the match. Cortisol increased by 78% (p = 0.103), whereas testosterone increased significantly by 44% (p = 0.004). No differences were seen from prematch to postmatch in the testosterone to cortisol ratio, immunoglobulin (Ig) A, IgM, or IgG. Sprinting is correlated with changes in CK and MYO and may therefore be associated with muscle damage in semiprofessional soccer players....

Nine games players (mean age 23.3 years, s = 2.8; height 1.73 m, s = 0.08; body mass 70.0 kg, s = 12.7) completed 14 laps of a measured circuit that incorporated intermittent running and directional changes, representative of the movements made by field hockey players during match-play. The distances and speeds recorded by a global positioning satellite (GPS) system (Spi Elite™) were compared statistically with speed measurements made using timing gates and distances measured using a calibrated trundle wheel, to establish the criterion validity of the GPS system. A validation of the speed of movement of each participant separately was also made, using data from each timing gate, over a range of speeds. The mean distance recorded by the GPS system was 6821 m (s = 7) and the mean speed was 7.0 km · h⁻¹ (s = 1.9), compared with the actual distance of 6818 m and recorded mean speed of 7.0 km · h⁻¹ (s = 1.9). Pearson correlations (r) among timing gate speed and GPS speed were ≥0.99 (P < 0.001) and the mean difference and 95% limits of agreement were 0.0 ± 0.9 km · h⁻¹. These results suggest that a GPS system (Spi Elite™) offers a valid tool for measuring speed and distance during match-play, and can quickly provide the scientist, coach, and player with objective information about certain movement patterns during competitive games.

Background: It is well documented that heat acclimation of six or more sessions of at least 60 min duration prolongs the time to exhaustion during endurance walking, cycling and running in the heat. However, this type of acclimation is not specific to team sport activity and the effect of acclimation on prolonged high-intensity intermittent running has not yet been investigated. Objective: To assess the impact of an intermittent acclimation protocol on distance run during team sport activity. Methods: The impact of four short heat acclimation sessions (30–45 min of the Loughborough Intermittent Shuttle Test; LIST) on high-intensity intermittent running capacity (LIST) in the heat (30°C, 27% relative humidity (RH)), was examined. Seventeen female well-trained games players were split into three groups: an acclimation group (30°C, 24% RH), a moderate training group (18°C, 41% RH) and a control group who did not complete any training between the main trials (pre-acclimation and post-acclimation). The pre-acclimation (A) and post-acclimation (B) trials were separated by 28 days to control for menstrual phase and verified using hormonal analysis. The four acclimation or moderate training sessions utilising the LIST were completed with one or two rest days interspersed between each session in a 10-day period prior to the post-acclimation trial (B). Results: In the post-acclimation trial distance run was increased by 33% in the acclimation group (A: 7703 (SEM 1401) m vs B: 10215 (SEM 1746) m; interaction group × trial p<0.05), but was unchanged in the moderate and control groups. The acclimation group had a lower rectal temperature (interaction group × trial × time p<0.01) due to a lower rate of rise, and an increase in thermal comfort1 after acclimation (End A: 7 (SEM 2) vs 6 (SEM 2); interaction group × trial p<0.01). There was no difference in serum progesterone, aldosterone or cortisol concentrations following acclimation or between groups. Conclusion: Four 30–45 min sessions of intermittent exercise induced acclimation, and resulted in an improvement in intermittent running exercise capacity in female games players. A lower rectal temperature and a concomitant rise in thermal comfort may be partly responsible for the improvement in exercise capacity.

We examined the effect of β-alanine supplementation plus sodium bicarbonate on high-intensity cycling capacity. Twenty males (age = 25 ± 5 yr, height = 1.79 ± 0.06 m, body mass = 80.0 ± 10.3 kg) were assigned to either a placebo (P) or a β-alanine (BA; 6.4 g·d(-1) for 4 wk) group based on power max, completing four cycling capacity tests at 110% of power max (CCT110%) to determine time to exhaustion (TTE) and total work done. A CCT(110%) was performed twice (habituation and baseline) before supplementation (with maltodextrin [MD]) and twice after supplementation (with MD and with sodium bicarbonate [SB]), using a crossover design with 2 d of rest between trials, creating four study conditions (PMD, PSB, BAMD, and BASB). Blood pH, Lactate, bicarbonate and base excess were determined at baseline, before exercise, immediately after exercise, and 5 min after exercise. Data were analyzed using repeated-measures ANOVA. TTE was increased in all conditions after supplementation (+1.6% PMD, +6.5% PSB, +12.1% BAMD, and +16.2% BASB). Both BAMD and BASB resulted in significantly improved TTE compared with that before supplementation (P ≤ 0.01). Although further increases in TTE (4.1%) were shown in BASB compared with BAMD, these differences were not significant (P = 0.74). Differences in total work done were similar to those of TTE. Blood bicarbonate concentrations were significantly (P ≤ 0.001) elevated before exercise in PSB and BASB but not in PMD or BAMD. Blood lactate concentrations were significantly elevated after exercise, remaining elevated after 5 min of recovery (P ≤ 0.001) and were highest in PSB and BASB. Results show that BA improved high-intensity cycling capacity. However, despite a 6-s (∼4%) increase in TTE with the addition of SB, this did not reach statistical significance, but magnitude-based inferences suggested a ∼70% probability of a meaningful positive difference.

Purpose: To determine whether gastrointestinal (GI) distress affects the ergogenicity of sodium bicarbonate and whether the degree of alkalemia or other metabolic responses is different between individuals who improve exercise capacity and those who do not.Methods: Twenty-one men completed 2 cycling-capacity tests at 110% of maximum power output. Participants were supplemented with 0.3 g/kg body mass of either placebo (maltodextrin) or sodium bicarbonate (SB). Blood pH, bicarbonate, base excess, and lactate were determined at baseline, preexercise, immediately postexercise, and 5 min postexercise.Results: SB supplementation did not significantly increase total work done (TWD; P = .16, 46.8 · 9.1 vs 45.6 · 8.4 kJ, d = 0.14), although magnitude-based inferences suggested a 63% likelihood of a positive effect. When data were analyzed without 4 participants who experienced GI discomfort, TWD (P = .01) was significantly improved with SB. Immediately postexercise blood lactate was higher in SB for the individuals who improved but not for those who did not. There were also differences in the preexercise-to-postexercise change in blood pH, bicarbonate, and base excess between individuals who improved and those who did not.Conclusions: SB improved high-intensity-cycling capacity but only with the exclusion of participants experiencing GI discomfort. Differences in blood responses suggest that SB may not be beneficial to all individuals. Magnitude-based inferences suggested that the exercise effects are unlikely to be negative; therefore, individuals should determine whether they respond well to SB supplementation before competition.