Drebrin A is a postsynaptic protein that localizes in vivo to the submembranous surface of dendritic sites forming excitatory synapses

Drebrin E is involved in the regulation of axonal growth through actin–myosin interactions

Activation of N-methyl-d-aspartate receptor induces a shift of drebrin distribution: Disappearance from dendritic spines and appearance in dendritic shafts

Genetic disruption of the alternative splicing of drebrin gene impairs context-dependent fear learning in adulthood

Evidence for cell density affecting C2C12 myogenesis: possible regulation of myogenesis by cell–cell communication

Drebrin a knockout eliminates the rapid form of homeostatic synaptic plasticity at excitatory synapses of intact adult cerebral cortex