Elizabeth Katherine Wood

Dr. Elizabeth Katherine Wood

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Research Focus & Metrics

Academic Career Length

9Years (2018-Present)

(Calculated once Scilit publication data is synced: current year − year of first publication + 1)

Representative Publications

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17 February 2023 in The Journal of Positive Psychology
Verified
Journal Article
Closed access
JRJuliette L. Ratchford
Juliette L. Ratchford
ACAmber R. Cazzell
Amber R. Cazzell
EWElizabeth Katherine Wood
Elizabeth Katherine Wood
BOBradley Owens
Bradley Owens
RQRobert Quinn
Robert Quinn
SSSarah Schnitker
Sarah Schnitker
In conceptualizing virtues as fully distinct constructs and vices as the absence of virtues, positive psychology is unable to provide a coherent account that explains tensions between virtues. This article puts forward a simple, holistic model that emphasizes the counterbalancing nature of virtues and conceptualizes vices as both deficiencies and excesses of virtues. Virtues check-and-balance one another to temper instantiations of vice. To test this model, a longitudinal study (N = 299) was conducted on goals and goal pursuit, investigating the virtuous strategies of patience and courage and their relations with the vices of passivity and reactivity. Findings suggested empirical support for the virtue counterbalancing model. Additionally, there were interesting distinctions at the within level (e.g., specific goal level) and between level (e.g., characteristic person level) of analysis, which provide further insight into how these virtues and vices relate. Implications for the model and future research directions are discussed.

1 August 2019 in Developmental Psychobiology
Verified
Journal Article
Closed access
PDF file
ABAlexander Baxter
Alexander Baxter
EWElizabeth Katherine Wood
Elizabeth Katherine Wood
LWLynea R. Witczak
Lynea R. Witczak
KBKaren L. Bales
Karen L. Bales
JHJ. Dee Higley
J. Dee Higley

11 March 2020 in Developmental Psychobiology
Verified
Journal Article
Closed access
PDF file
ABAshley M. Sproul Bassett
Ashley M. Sproul Bassett
EWElizabeth Katherine Wood
Elizabeth Katherine Wood
SLStephen G. Lindell
Stephen G. Lindell
MSMelanie L. Schwandt
Melanie L. Schwandt
CBChristina S. Barr
Christina S. Barr
JHJames D. Higley
James D. Higley

1 November 2021 in Journal of the American Academy of Child & Adolescent Psychiatry
Verified
Journal Article
Closed access
EWElizabeth Katherine Wood
Elizabeth Katherine Wood
WEWhitney F. Espinel
Whitney F. Espinel
JHJacob Hunter
Jacob Hunter
AEAlexa Emmett
Alexa Emmett
ASAndrea N. Skowbo
Andrea N. Skowbo
JHJ. Dee Higley
J. Dee Higley

14 April 2021 in Neuropsychopharmacology
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Journal Article
Closed access
PDF file
EWElizabeth Katherine Wood
Elizabeth Katherine Wood
RKRyno Kruger
Ryno Kruger
JDJaclyn P. Day
Jaclyn P. Day
SDStephen M. Day
Stephen M. Day
JHJacob N. Hunter
Jacob N. Hunter
JHJ. Dee Higley
J. Dee Higley

4 February 2019 in Development and Psychopathology
Verified
Journal Article
Closed access
PDF file
ABAlexander Baxter
Alexander Baxter
EWElizabeth Katherine Wood
Elizabeth Katherine Wood
CBChristina S. Barr
Christina S. Barr
DKDaniel B. Kay
Daniel B. Kay
SSStephen J. Suomi
Stephen J. Suomi
JHJ. Dee Higley
J. Dee Higley
Environmental and biological factors contribute to sleep development during infancy. Parenting plays a particularly important role in modulating infant sleep, potentially via the serotonin system, which is itself involved in regulating infant sleep. We hypothesized that maternal neglect and serotonin system dysregulation would be associated with daytime sleep in infant rhesus monkeys. Subjects were nursery-reared infant rhesus macaques (n = 287). During the first month of life, daytime sleep-wake states were rated bihourly (0800–2100). Infants were considered neglected (n = 16) if before nursery-rearing, their mother repeatedly failed to retrieve them. Serotonin transporter genotype and concentrations of cerebrospinal fluid 5-hydroxyindoleacetic acid (5-HIAA) were used as markers of central serotonin system functioning. t tests showed that neglected infants were observed sleeping less frequently, weighed less, and had higher 5-HIAA than non-neglected nursery-reared infants. Regression revealed that serotonin transporter genotype moderated the relationship between 5-HIAA and daytime sleep: in subjects possessing the Ls genotype, there was a positive correlation between 5-HIAA and daytime sleep, whereas in subjects possessing the LL genotype there was no association. These results highlight the pivotal roles that parents and the serotonin system play in sleep development. Daytime sleep alterations observed in neglected infants may partially derive from serotonin system dysregulation.