Numerous cross‐sectional studies have attempted to identify the muscle morphology required to achieve high sprint velocity. Our longitudinal study addressed an unanswered question of cross‐sectional studies: whether hypertrophy of the individual trunk and thigh muscles induced by daily training (e.g., sprint, jump, and resistance training) is linked to an improvement in sprint performance within well‐trained sprinters. Twenty‐three collegiate male sprinters (100‐m best time of 11.36 ± 0.44 s) completed their daily training for 1 year without our intervention. Before and after the observation period, the sprint velocities at 0–100 m, 0–10 m, and 50–60 m intervals were measured using timing gates. The volumes of 14 trunk and thigh muscles were measured using magnetic resonance imaging. Muscle volumes were normalized to the participants' body mass at each time point. Sprint velocities increased at the 0–100 m (p < 0.001), 0–10 m (p = 0.019), and 50–60 m (p = 0.018) intervals after the observation period. The relative volumes of the tensor fasciae latae, sartorius, biceps femoris long head, biceps femoris short head, semitendinosus, and iliacus were increased (all p < 0.050). Among the hypertrophied muscles, only the change in the relative volume of the semitendinosus was positively correlated with the change in sprint velocity at the 50–60 m interval (p = 0.018 and ρ = 0.591). These findings suggest that semitendinosus hypertrophy seems to be associated with sprint performance improvement within well‐trained sprinters during the maximal velocity phase.

We investigated effects of molecular hydrogen (H₂) supplementation on acid-base status, pulmonary gas exchange responses, and local muscle oxygenation during incremental exercise. Eighteen healthy, trained subjects in a randomized, double-blind, crossover design received H₂-rich calcium powder (HCP) (1500 mg/day, containing 2.544 µg/day of H₂) or H₂-depleted placebo (1500 mg/day) for three consecutive days. They performed cycling incremental exercise starting at 20-watt work rate, increasing by 20 watts/2 min until exhaustion. Breath-by-breath pulmonary ventilation ($\dot{\mathrm{V}}$_E) and CO₂ output ($\dot{\mathrm{V}}$CO₂) were measured and muscle deoxygenation (deoxy[Hb + Mb]) was determined via time-resolved near-infrared spectroscopy in the vastus lateralis (VL) and rectus femoris (RF). Blood gases’ pH, lactate, and bicarbonate (HCO₃⁻) concentrations were measured at rest and 120-, 200-, and 240-watt work rates. At rest, the HCP group had significantly lower $\dot{\mathrm{V}}$_E, $\dot{\mathrm{V}}$CO₂, and higher HCO₃⁻, partial pressures of CO₂ (PCO₂) versus placebo. During exercise, a significant pH decrease and greater HCO₃⁻ continued until 240-watt workload in HCP. The $\dot{\mathrm{V}}$_E was significantly lower in HCP versus placebo, but HCP did not affect the gas exchange status of $\dot{\mathrm{V}}$CO₂ or oxygen uptake ($\dot{\mathrm{V}}$O₂). HCP increased absolute values of deoxy[Hb + Mb] at the RF but not VL. Thus, HCP-induced hypoventilation would lead to lower pH and secondarily impaired balance between O₂ delivery and utilization in the local RF during exercise, suggesting that HCP supplementation, which increases the at-rest antioxidant potential, affects the lower ventilation and pH status during incremental exercise. HPC induced a significantly lower O₂ delivery/utilization ratio in the RF but not the VL, which may be because these regions possess inherently different vascular/metabolic control properties, perhaps related to fiber-type composition.

This research examined the effects of single-dose molecular hydrogen (H₂) supplements on acid-base status and local muscle deoxygenation during rest, high-intensity intermittent training (HIIT) performance, and recovery. Ten healthy, trained subjects in a randomized, double-blind, crossover design received H₂-rich calcium powder (HCP) (1500 mg, containing 2.544 μg of H₂) or H₂-depleted placebo (1500 mg) supplements 1 h pre-exercise. They performed six bouts of 7 s all-out pedaling (HIIT) at 7.5% of body weight separated by 40 s pedaling intervals, followed by a recovery period. Blood gases’ pH, PCO₂, and HCO₃⁻ concentrations were measured at rest. Muscle deoxygenation (deoxy[Hb + Mb]) and tissue O₂ saturation (S_tO₂) were determined via time-resolved near-infrared spectroscopy in the vastus lateralis (VL) and rectus femoris (RF) muscles from rest to recovery. At rest, the HCP group had significantly higher PCO₂ and HCO₃⁻ concentrations and a slight tendency toward acidosis. During exercise, the first HIIT bout’s peak power was significantly higher in HCP (839 ± 112 W) vs. Placebo (816 ± 108 W, p = 0.001), and HCP had a notable effect on significantly increased deoxy[Hb + Mb] concentration during HIIT exercise, despite no differences in heart rate response. The HCP group showed significantly greater O₂ extraction in VL and microvascular (Hb) volume in RF during HIIT exercise. The HIIT exercise provided significantly improved blood flow and muscle reoxygenation rates in both the RF and VL during passive recovery compared to rest in all groups. The HCP supplement might exert ergogenic effects on high-intensity exercise and prove advantageous for improving anaerobic HIIT exercise performance.

The findings of previous studies support the efficacy of cold water immersion (CWI) with carbon dioxide (CO₂) in enhancing muscle blood flow and maintaining aerobic performance efficiency. We hypothesize that the addition of hydrogen gas (H₂), known for its antioxidant properties and role in inflammation regulation, to C-CWI can enhance recovery after eccentric exercise. and Methods: Thirty-four healthy subjects performed a knee-extensor eccentric exercise. They were randomly allocated into four groups: control, CWI, CO₂-rich CWI (C-CWI), and CO₂ + H₂ gas mixture CWI (CH-CWI). In the three CWI groups, all subjects were immersed in the appropriate bath at 20 °C for 20 min immediately after 60 repetitions of eccentric exercise. Before exercise and after 48 h of recovery, the subjects' maximal voluntary isometric contraction torque (MVC-ISO), maximal voluntary concentric (MVC-CON) contraction torque, countermovement jump (CMJ) height, knee flexion range of motion (ROM), muscle soreness, and muscle thickness were measured. In the CH-CWI group only, the MVC-ISO, CMJ height, and ROM did not decrease significantly post-exercise, whereas all of these decreased in the other three groups. Muscle soreness at palpation, contraction, and stretching significantly increased post-exercise in all groups. Echo intensity and tissue hardness did not increase significantly in the CH-CWI group. CH-CWI stimulated recovery from impairments in MVC-ISO torque, CMJ height, knee-flexion ROM, tissue hardness, and echo intensity. These findings indicate that CH-CWI can promote recovery after eccentric exercise.

We investigated cardiovascular responses to an orthostatic challenge in trained spinal cord-injured (SCI) individuals compared to able-bodied (AB) individuals. A total of 23 subjects participated, divided into three groups: seven were trained as spinal cord-injured (Tr-SCI) individuals, seven were able-bodied individuals trained as runners (Tr-AB), and nine were untrained able-bodied individuals (UnTr-AB). We measured the cardiovascular autonomic responses in all three groups during each 5-min head-up tilt (HUT) of 0°, 40°, and 80°. Stroke volume (SV), heart rate (HR), and cardiac output (Qc) as cardiovascular responses were measured by impedance cardiography. Changes in deoxyhemoglobin (∆[HHb]) and total hemoglobin (∆[Hbtot]) concentrations of the right medial gastrocnemius muscle were measured using near-infrared spectroscopy (NIRS). As the HUT increased from 0° to 80°, Tr-SCI group showed less change in SV at all HUT levels even if HR increased significantly. Mean arterial pressure (MAP) also did not significantly increase as tilting increased from 0° to 80°. Regarding peripheral vascular responses, the alterations of ∆[Hbtot] from 0° to 80° were less in Tr-SCI group compared to AB individuals. There is a specific mechanism whereby blood pressure is maintained during a HUT in Tr-SCI group with the elicitation of peripheral vasoconstriction and the atrophy of the vascular vessels in paraplegic lower limbs, which would be associated with less change in SV in response to an orthostatic challenge.

Side-To-Side Difference in Electromyographic Activity of Abdominal Muscles during Asymmetric Exercises