Prof. Giancarlo Panzica
Univ Turin, Dept Neurosci, Rita Levi Montalcini, I-10126 Turin, Italy
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Academic Career Length
52Years (1975-Present)
(Calculated once Scilit publication data is synced: current year − year of first publication + 1)
Representative Publications
View All Publications1 March 2017 in Reproductive Toxicology
Journal Article
JHJerrold J. Heindel
Jerrold J. HeindelBBBruce Blumberg
Bruce Blumberg
MCMathew Cave
Mathew Cave
RMRonit Machtinger
Ronit Machtinger
AMAlberto Mantovani
Alberto Mantovani
FSFrederick Vom Saal
Frederick Vom Saal
+6
The recent epidemics of metabolic diseases, obesity, type 2 diabetes(T2D), liver lipid disorders and metabolic syndrome have largely been attributed to genetic background and changes in diet, exercise and aging. However, there is now considerable evidence that other environmental factors may contribute to the rapid increase in the incidence of these metabolic diseases. This review will examine changes to the incidence of obesity, T2D and non-alcoholic fatty liver disease (NAFLD), the contribution of genetics to these disorders and describe the role of the endocrine system in these metabolic disorders. It will then specifically focus on the role of endocrine disrupting chemicals (EDCs) in the etiology of obesity, T2D and NAFLD while finally integrating the information on EDCs on multiple metabolic disorders that could lead to metabolic syndrome. We will specifically examine evidence linking EDC exposures during critical periods of development with metabolic diseases that manifest later in life and across generations.
27 September 2011 in Journal of Neuroendocrinology
Journal Article
Cheryl Anne FryeEBE. Bo
E. Bo
Gemma CalamandreiLCLaura Calzà
Laura CalzàFDF. Dessì‐Fulgheri
F. Dessì‐FulgheriGiancarlo Panzica +8
1 March 2006 in Neuroscience
Journal Article
GVG.J. De Vries
G.J. De VriesGiancarlo Panzica1 January 1996 in Frontiers in Neuroendocrinology
Journal Article
Giancarlo PanzicaCVCarla Viglietti-Panzica
Carla Viglietti-Panzica
JBJacques Balthazartb
Jacques Balthazartb
GCGian CarloPanzicaa
Gian CarloPanzicaa
Journal Article
Maria E Street
Sabrina AngeliniSBSergio Bernasconi
Sergio BernasconiEBErnesto Burgio
Ernesto BurgioACAlessandra Cassio
Alessandra CassioSASergio Amarri
Sergio Amarri +18
Wildlife has often presented and suggested the effects of endocrine disrupting chemicals (EDCs). Animal studies have given us an important opportunity to understand the mechanisms of action of many chemicals on the endocrine system and on neurodevelopment and behaviour, and to evaluate the effects of doses, time and duration of exposure. Although results are sometimes conflicting because of confounding factors, epidemiological studies in humans suggest effects of EDCs on prenatal growth, thyroid function, glucose metabolism and obesity, puberty, fertility, and on carcinogenesis mainly through epigenetic mechanisms. This manuscript reviews the reports of a multidisciplinary national meeting on this topic.
20 June 2015 in Environmental Health
Journal Article
JHJerrold J. Heindel
Jerrold J. HeindelFSFrederick S. Vom Saal
Frederick S. Vom SaalBBBruce Blumberg
Bruce BlumbergPBPatrizia Bovolin
Patrizia BovolinGemma Calamandrei
Paola Palanza +25
A multidisciplinary group of experts gathered in Parma Italy for a workshop hosted by the University of Parma, May 16–18, 2014 to address concerns about the potential relationship between environmental metabolic disrupting chemicals, obesity and related metabolic disorders. The objectives of the workshop were to: 1. Review findings related to the role of environmental chemicals, referred to as “metabolic disruptors”, in obesity and metabolic syndrome with special attention to recent discoveries from animal model and epidemiology studies; 2. Identify conclusions that could be drawn with confidence from existing animal and human data; 3. Develop predictions based on current data; and 4. Identify critical knowledge gaps and areas of uncertainty. The consensus statements are intended to aid in expanding understanding of the role of metabolic disruptors in the obesity and metabolic disease epidemics, to move the field forward by assessing the current state of the science and to identify research needs on the role of environmental chemical exposures in these diseases. We propose broadening the definition of obesogens to that of metabolic disruptors, to encompass chemicals that play a role in altered susceptibility to obesity, diabetes and related metabolic disorders including metabolic syndrome.
About Me
Giancarlo Panzica (Ph.D. Honorary Professor, the Department of Neuroscience, University of Torino, Italy) obtained a master’s degree in biology at the University of Torino and earned his PhD degree at the Department of Anatomy, University of Salamanca (Spain). He had a long academic career at the University of Torino, starting as Researcher and then Associate Professor, and finally Full Professor in Human Anatomy. During this period, he was Director of the Department of Anatomy, Pharmacology and Forensic Medicine, and Director of the Department of Neuroscience. Prof. Panzica is the President of the Italian Group for the Study of Neuromorphology. He is currently retired from the University, but still active in research as Principal Investigator of the Neuroendocrinology laboratory at the Neuroscience Institute Cavalieri-Ottolenghi (NICO), Orbassano (Italy). Giancarlo Panzica is the author of 199 publications listed in PubMed and has an H-index of 41 (Scopus) with more than 5,900 citations.
The main focus of his research is the interaction of steroid hormones and the nervous system and its impact on physiology, behaviour, and neural diseases. In the last period, prof. Panzica has explored the permanent or transient effects of exposure to endocrine disruptors on the nervous system, particularly on those neuroendocrine circuits involved in the control of reproduction.