Objective: The objective of this article is to evaluate the clinical characteristics of spontaneous intracranial hemorrhages occurring in the post–COVID-19 (post-coronavirus disease 2019) period in patients without classical risk factors (hypertension, anticoagulant therapy, or vascular malformations) and to assess the concordance of these findings with the existing literature. Materials and Methods: Between 2020 and 2024, 36 patients with a prior history of COVID-19 who presented with spontaneous intracerebral hemorrhage were retrospectively reviewed. Cases were evaluated for age, sex, Glasgow Coma Scale (GCS), international normalized ratio (INR), hemorrhage type and anatomical location, modified Rankin Scale (mRS), rebleeding, and overall clinical outcomes. Patients with any macroscopic vascular pathology were excluded from the study. Results: The mean age of the patients was 52.0 years, and 61.1% were male. The overall mortality rate was 47.2%. Parenchymal hemorrhages comprised 55.6% of cases, subarachnoid hemorrhages 22.2%, and subdural hemorrhages 22.2%. Subarachnoid hemorrhages exhibited the highest mortality rate (87.5%). The mean GCS score was 8.2, and the mean modified Rankin Scale score at one year was 4.3. The GCS score was significantly associated with mortality (p=0.0001), whereas INR and age showed no significant relationship (p=0.2923 and p=0.6003, respectively). Conclusion: In the post–COVID-19 period, intracerebral hemorrhages occurring in patients without classical etiological factors represent a notable clinical entity. The high mortality and rates of functional dependency observed indicate that this patient group requires novel strategies for early diagnosis and long-term monitoring.

Introduction: Osteopetrosis is an inherited disease that causes thickening of the cranial bone. The need for both cerebral decompression and intracranial volume-enlarging cranioplasty in the same surgical session in a patient whose intracranial pressure is elevated cannot be prevented and is a very rare decision in neurosurgery practice. It is seen in the literature that the only indication that can lead to this surgery is osteopetrosis. Case report: A 24-year-old female patient with osteopetrosis admitted with the complaint of severe headache that did not respond to medical treatment, and surgery was decided. After decompressive craniectomy was performed with difficulties, cranioplasty with titanium mesh was performed in the same session to protect the cerebral tissue, to close the defect area, and to give more space to the cerebral tissue. Conclusions: It was observed that the complaints of increased intracranial pressure were completely resolved with unilateral cranial decompression in the early postoperative period and at the end of 2 years. The surgical intervention technique performed on the patient and the radiological findings obtained during the follow-up are presented. We think that performing cranioplasty with titanium mesh, which is used to protect and save the cerebral tissue in patients with osteopetrosis, is the most appropriate choice.

Bir sonraki kitabınız ne zaman çıkacak? Denildiğinde ihtiyaçlara cevap veren kitaplara öncülük etmeye başladığımızı anladım. Bu kitap projesi ile yaptığımız, kliniklerde çalışan hekimler olarak ihtiyaçların gün yüzüne çıkardığı en ufak ayrıntılar ve bunlarla başa çıkmak için sarf ettiğimiz gayretlerin kitaba dönüştürülmesi oldu. Genel olarak yazılan radyasyon onkoloji kitaplarının aksine bu kitap; iyonize radyasyonun etkileri kadar, radyoterapinin yan etkilerinin çözümünde multidisipliner yaklaşım gereksiniminden yola çıkarak neredeyse tüm disiplinlerin katılımı ile yazıldı. Radyasyona dahil temel konular, radyasyonun patolojisi, radyasyon radyolojisi, akut radyasyon maruziyeti, radyoterapi sonrası cerrahi zorlukları ve radyasyondan korunma ana bölümler altında seçilmiş konular anlatıldı.

Considerable interindividual variability exists in the spread and duration of spinal anesthesia despite standardized anesthetic techniques. Lumbosacral cer

A 54-year-old male with a previously diagnosed vertebrobasilar aneurysm remained asymptomatic for 8 years, during which the aneurysm was believed to have spontaneously thrombosed. He later developed progressive gait disturbance and hydrocephalus, but notably, no dysphagia. Following ventriculoperitoneal shunting, the sudden reduction in cerebrospinal fluid (CSF) eliminated the protective buffer around the brainstem, leading to direct aneurysmal pulsations and the abrupt onset of dysphagia. This case highlights the critical role of CSF in masking brainstem compression symptoms and underscores the risk of rapid neurological decline following abrupt CSF depletion. Careful CSF management is essential in patients with hydrocephalus and large posterior circulation aneurysms to prevent severe complications.